Evidence map›Paper›PMID 41155471›Full record

ArticleInternational journal of molecular sciences2025

Developing Endogenous Autophagy Reporters in

Kincső Bördén, Tibor Vellai, Tímea Sigmond

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in International journal of molecular sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Autophagy: A Double-Edged Sword in the Aging ofInternational journal of molecular sciences · 2026
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Kincső BördénDepartment of Genetics, Eötvös Loránd University (ELTE), Pázmány Péter sétány 1/C, 1117 Budapest, Hungary.
Tibor VellaiDepartment of Genetics, Eötvös Loránd University (ELTE), Pázmány Péter sétány 1/C, 1117 Budapest, Hungary.
Tímea SigmondDepartment of Genetics, Eötvös Loránd University (ELTE), Pázmány Péter sétány 1/C, 1117 Budapest, Hungary.ORCID 0000-0002-1985-428X

Funding

Enhancing and expanding the CGC Strain CollectionP40OD010440 · OD · UNIVERSITY OF MINNESOTA · PI Ann E. Rougvie · 2012 to 2026
$7.5M
NIH HHS P40 OD010440OTKA K132439
6 · The paper itself

Abstract

Autophagy (cellular self-eating) is a tightly regulated catabolic process of eukaryotic cells during which parts of the cytoplasm are sequestered and subsequently delivered into lysosomes for degradation by acidic hydrolases. This process is central to maintaining cellular homeostasis, the removal of aged or damaged organelles, and the elimination of intracellular pathogens. The nematode

Indexed as

AutophagyCaenorhabditis elegansGenes, ReporterStarvationAnimalsAnimals, Genetically ModifiedCaenorhabditis elegans ProteinsGreen Fluorescent ProteinsLuminescent ProteinsLysosomesMicrotubule-Associated ProteinsRed Fluorescent ProteinCaenorhabditis elegans ProteinsGreen Fluorescent ProteinsLGG-1 protein, C elegansLuminescent ProteinsMicrotubule-Associated ProteinsRed Fluorescent ProteinATG-5autophagyC. elegansendogenous reportersLGG-1starvationTOR

Identifiers

PMID41155471
PMCPMC12563416

What OpenQuestion holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.