ArticleNature communications2025
A circadian morning complex modulates far-red light signaling in Arabidopsis.
Article in Nature communications, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
4 citing papers in PubMed.
- Natural variation in GmRVE4d facilitates soybean latitudinal adaptation by regulating GmPRR5a-dependent flowering and maturity.Nature communications · 2026Article
- Light at the heart of time: How photoreceptors entrain the circadian clock.Plant communications · 2026Review
- Review
- RrLHY regulates arginine biosynthesis by activating RrNAGS1 in Rosa roxburghii fruit.Plant cell reports · 2026Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
7 authors.
Funding
Abstract
The plant circadian clock coordinates internal processes with daily and seasonal environmental changes by interacting with prevalent light cues. However, how the circadian clock feedback regulates light signals remains largely elusive. Here, we identify that the clock regulator TIME FOR COFFEE (TIC), which interacts with the core clock components CIRCADIAN CLOCK ASSOCIATED1 (CCA1) and LATE ELONGATED HYPOCOTYL (LHY) to form a "Morning Complex" in the nucleus and co-repress numerous genes, particularly at dawn. Intriguingly, the MC inhibits PHYA and other phyA signaling components at dawn through binding to their promoters. Mutants lacking CCA1 and LHY show increased sensitivity to far-red light, similar to tic mutants, highlighting the cooperative role of TIC, CCA1, and LHY in regulating light-inhibited hypocotyl growth. Altogether, these findings indicate that the circadian MC is a key complex, feedback regulates light signal and mediates multiple biological processes at dawn to ensure plant fitness.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.