Evidence map›Paper›PMID 41145827›Full record

ArticleEuropean journal of human genetics : EJHG2026

Heterozygous loss of SRRM1 may be associated with neurodevelopmental phenotypes and anomalies in cell growth and neurite morphology.

Melek Firat Altay, Anne Gregor, Dominique Braun, Claudine Rieubland, Matthias Gautschi, Eveline Perret Hoigné, Rike Schiller, Boris Keren, Alejandra Afenjar, Undiagnosed Diseases Network and 3 more

Abstract read
In one paragraph

Article in European journal of human genetics : EJHG, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors.

Melek Firat AltayDepartment of Human Genetics, Inselspital, University of Bern, Bern, Switzerland.ORCID http://orcid.org/0000-0002-8174-5631
Anne GregorDepartment of Human Genetics, Inselspital, University of Bern, Bern, Switzerland.
Dominique BraunDepartment of Human Genetics, Inselspital, University of Bern, Bern, Switzerland.
Claudine RieublandDepartment of Human Genetics, Inselspital, University of Bern, Bern, Switzerland.
Matthias GautschiDivision of Paediatric Endocrinology, Diabetology and Metabolism, Department of Paediatrics, and Institute of Clinical Chemistry, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.ORCID http://orcid.org/0000-0003-1358-1759
Eveline Perret HoignéDivision of Neuropediatrics, Department of Paediatrics, Inselspital, Bern University Hospital, Bern, Switzerland.
Rike SchillerDepartment of Diagnostic, Interventional and Paediatric Radiology, University of Bern, Inselspital Universitätsspital Bern, Bern, Switzerland.
Boris KerenAPHP, Sorbonne Université, Département de génétique médicale, GH Pitié Salpêtrière, Paris, France.
Alejandra AfenjarClinical Genetics Unit, Reference Center for Cerebellar Malformations and Congenital Diseases, Armand-Trousseau Hospital, APHP, Sorbonne University, Paris, France.
Undiagnosed Diseases Network
Julian A Martinez-AgostoDepartment of Human Genetics, UCLA, Los Angeles, CA, USA.
Jill A RosenfeldDepartment of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX, USA.ORCID http://orcid.org/0000-0001-5664-7987
Christiane ZweierDepartment of Human Genetics, Inselspital, University of Bern, Bern, Switzerland. christiane.zweier@insel.ch.ORCID http://orcid.org/0000-0001-8002-2020

Funding

Diagnosing the Unknown for Care and Advancing Science (DUCAS)U2CNS132415 · NINDS · HARVARD MEDICAL SCHOOL · PI Francis Sessions Cole · 2023 to 2026
$32.1M
Clinical Sequencing Core Facility for the Undiagnosed Diseases Network (UDN)U01HG007942 · NHGRI · BAYLOR COLLEGE OF MEDICINE · PI ENG, CHRISTINE · 2014 to 2021
$10.2M
UCLA clinical site for the investigation of undiagnosed disordersU01NS134356 · NINDS · UNIVERSITY OF CALIFORNIA LOS ANGELES · PI MARTINEZ-AGOSTO, JULIAN, NELSON, STANLEY F. · 2023 to 2023
$936k
Deutsche Forschungsgemeinschaft (German Research Foundation) ZW184/6-1NHGRI NIH HHS U01 HG007942NINDS NIH HHS U01 NS134356NINDS NIH HHS U2C NS132415Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung (Swiss National Science Foundation) 10001220U.S. Department of Health & Human Services | NIH | National Institute of Neurological Disorders and Stroke (NINDS) U01HG007942U.S. Department of Health & Human Services | NIH | National Institute of Neurological Disorders and Stroke (NINDS) U01NS134356U.S. Department of Health & Human Services | NIH | National Institute of Neurological Disorders and Stroke (NINDS) U2CNS132415
6 · The paper itself

Abstract

Serine/arginine repetitive matrix protein 1 (SRRM1) is a key component of spliceosomes and plays various roles in messenger RNA processing. To date, its function in the nervous system has not been elucidated, and germline variants in SRRM1 have not yet been implicated in disease. Through international collaboration, we have identified three individuals harbouring heterozygous truncating variants in SRRM1, presenting variably with developmental delay, intellectual disability, short stature, behavioural and skeletal anomalies, and facial dysmorphism. Two of the variants occurred de novo, while the third could not be tested in the parents. Reduction of SRRM1 to 50% in SKNBE2 cells by introducing a truncating variant via CRISPR-Cas9 editing, followed by differentiation into neuron-like cells, resulted in impaired cell proliferation, migration, and neurite outgrowth compared to wild-type cells. Additionally, the role of SRRM1 in nervous system development and functioning was investigated in vivo using a Drosophila model. Pan-neuronal knockdown of the orthologue Srrm1 led to reduced viability, while motoneuronal knockdown impaired gross neurological function. Taken together, we provide multiple lines of evidence that loss of SRRM1 is associated with nervous system-related phenotypes, and that its haploinsufficiency may be causative for a neurodevelopmental disorder.

Indexed as

Developmental DisabilitiesNeuritesNeurodevelopmental DisordersAnimalsCell ProliferationChildChild, PreschoolFemaleHaploinsufficiencyHeterozygoteHumansIntellectual DisabilityMalePhenotype

Identifiers

PMID41145827
PMCPMC12858978

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.