ReviewImmunology and allergy clinics of North America2025
The Mechanisms of Immunoglobulin E Sensitizations in Atopic Dermatitis.
Review in Immunology and allergy clinics of North America, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
3 citing papers in PubMed.
- Assessment of Common Food Allergens in Infants With Atopic Dermatitis in Kashan From 2005 to 2021.Immunity, inflammation and disease · 2026Article
- Basophils in atopic dermatitis: Immunologic roles and clinical implications.Annals of allergy, asthma & immunology : official publication of the American College of Allergy, Asthma, & Immunology · 2026Review
- Omalizumab in Refractory Adult-Onset Facial Atopic Dermatitis: A Case Report.Clinical, cosmetic and investigational dermatology · 2026Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
Abstract
Atopic dermatitis (AD) is a chronic inflammatory skin disorder, which often precedes food allergy, asthma, and allergic rhinitis-a progression known as the atopic march. A key feature of AD is elevated allergen-specific immunoglobulin E (IgE), indicating sensitization to environmental, microbial, and self-antigens. This study examines the mechanisms driving IgE sensitization in AD, including epidermal barrier dysfunction, immune dysregulation, antigen presentation, microbial imbalance, and genetic susceptibility. Understanding these pathways is essential for developing targeted therapies to prevent allergic disease progression and improve clinical outcomes.
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Registered trials
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