Evidence map›Paper›PMID 41131368›Full record

ReviewCurrent nutrition reports2025

Zinc and Adipose Organ Dysfunction: Molecular Insights into Obesity and Metabolic Disorders.

Zahra Bahadoran, Fateme Ghafouri-Taleghani, Marijana Todorčević

Abstract readReview
PubMed Publisher
In one paragraph

Review in Current nutrition reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. Review
  2. Review
  3. Article
  4. Article
  5. Review
  6. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Zahra BahadoranMicronutrient Research Center, Research Institute for Endocrine Disorders, Research Institute for Endocrine Sciences, Shahid Beheshti University of Medical Sciences, Tehran, Iran. zahrabahadorabn@yahoo.com.
Fateme Ghafouri-TaleghaniMicronutrient Research Center, Research Institute for Endocrine Disorders, Research Institute for Endocrine Sciences, Shahid Beheshti University of Medical Sciences, Tehran, Iran.
Marijana TodorčevićDepartment of Biochemistry and Physiology, School of Pharmacy and Food Sciences, Institut de Biomedicina de la Universitat de Barcelona (IBUB), Universitat de Barcelona, Barcelona, E-08028, Spain.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

purpose of reviewThis review explores the emerging role of zinc (Zn) as a critical regulator of adipose organ structure and function, highlighting how Zn-related molecular networks contribute to the pathogenesis of obesity and associated metabolic disorders. RECENT

findingsExperimental Zn-deficient models, and genetically-modified Zn-associated proteins, including Zn transporters [i.e., ZnTs (SLC30 family) and Zips (SLC39 family)], zinc-α

Indexed as

Adipose TissueMetabolic DiseasesObesityZincAdipocytesAdipogenesisAnimalsCarrier ProteinsCation Transport ProteinsHumansMetallothioneinCarrier ProteinsCation Transport ProteinsMetallothioneinZincAdipose organInsulin resistanceObesitySubcutaneous adiposeVisceral adiposeZinc

Identifiers

PMID41131368

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.