ReviewCancer cell international2025
Ferroptosis and non-coding RNAs in lung cancer: exploring molecular mechanisms and diagnostic/therapeutic implications.
Review in Cancer cell international, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
2 citing papers in PubMed.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Lung cancer is the second most prevalent type of cancer on a global scale, and is widely known for being the deadliest. Revealing the fundamental molecular mechanisms of this condition has the potential to unlock fresh possibilities and avenues for managing it. Ferroptosis is a newly discovered type of controlled cell death that distinguishes itself from traditional forms of programmed cell death, specifically apoptosis and necrosis, through its distinct biochemical and structural characteristics. Changes made to the mechanism that regulates ferroptosis can have a significant influence on the development of various illnesses, such as, though not limited to, lung cancer. Based on scientific studies, it has been proven that cancer cells possess a significant capability to inhibit ferroptosis, leading to their continuous growth and survival. The presence of non-coding RNAs has a profound influence on controlling a wide range of cellular functions, with a particular focus on cancer. They have been closely associated with all key features of lung cancer. Fresh findings have indicated that these particles also have the task of controlling ferroptosis. Thus, the use of non-coding RNA as a therapeutic strategy offers a potentially effective option for regulating ferroptosis in the management of cancer. The main focus of this overview is on ncRNAs and their function in controlling ferroptosis, specifically within the context of lung cancer.
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