ArticleFrontiers in cellular neuroscience2025
Sex-specific interneuron vulnerability after traumatic brain injury correlates with neurotrophic signaling and chloride homeostasis, independent of behavioral and network outcomes.
Article in Frontiers in cellular neuroscience, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
2 citing papers in PubMed.
- Association Between Hyperchloremia and Neurological Outcomes in Traumatic Brain Injury: A Narrative Review.Healthcare (Basel, Switzerland) · 2026Review
- Analyzing the Spectrum of mTBI Subgroups.Neurotrauma reportsReview
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Authors and funding
7 authors.
Funding
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Abstract
Traumatic brain injury (TBI) leads to persistent cognitive and emotional impairments, and growing evidence suggests that sex influences vulnerability through differences in neurotrophic signaling and chloride homeostasis. To investigate these mechanisms, we induced moderate TBI in male and female mice using the controlled cortical impact model and assessed outcomes 30 days post-injury. Behavioral performance was evaluated with the open field, elevated plus maze, and Barnes maze, while hippocampal oscillations, interneuron survival, protein expression (KCC2, NKCC1, p75
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Registered trials
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