Article in American journal of physiology. Endocrinology and metabolism, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Liam S FitzgeraldDepartment of Orthopaedic Surgery, School of Medicine, University of California San Diego, La Jolla, California, United States.ORCID 0000-0001-8421-6383
Connor S Reynoso SpurrierDepartment of Orthopaedic Surgery, School of Medicine, University of California San Diego, La Jolla, California, United States.ORCID 0009-0008-6222-3839
Nathan J LauDepartment of Orthopaedic Surgery, School of Medicine, University of California San Diego, La Jolla, California, United States.
Miles D MelamedDepartment of Orthopaedic Surgery, School of Medicine, University of California San Diego, La Jolla, California, United States.
Lindsey A BurnettDepartment of Obstetrics, Gynecology, and Reproductive Sciences, School of Medicine, University of California San Diego, La Jolla, California, United States.ORCID 0000-0002-6430-4498
Gretchen A MeyerProgram in Physical Therapy, Washington University School of Medicine, St. Louis, Missouri, United States.ORCID 0000-0001-9268-3993
Chang GuiDepartment of Biomedical Engineering, Washington University in St. Louis, St. Louis, Missouri, United States.
Andrea L HevenerDepartment of Medicine, University of California Los Angeles, Los Angeles, California, United States.ORCID 0000-0003-1508-4377
James A SanfordBiological Sciences Division, Pacific Northwest National Laboratory, Richland, Washington, United States.ORCID 0000-0001-7901-5579
Simon SchenkDepartment of Orthopaedic Surgery, School of Medicine, University of California San Diego, La Jolla, California, United States.ORCID 0000-0002-8224-3203
Funding
Transgenic & Knock-out MouseP30DK063491 · NIDDK · UNIVERSITY OF CALIFORNIA, SAN DIEGO · PI MILES Frome WILKINSON · 2003 to 2026
$40.4M
REPRODUCTIVE SCIENTIST TRAINING PROGRAMK12HD000849 · NICHD · WASHINGTON UNIVERSITY · PI Danny J Schust · 1988 to 2026
$33.2M
MEDICAL SCIENTIST TRAINING PROGRAMT32GM007198 · NIGMS · UNIVERSITY OF CALIFORNIA, SAN DIEGO · PI CHI, NEIL C, INSEL, PAUL A · 1985 to 2024
$29.3M
Sex Differences in the Metabolic SyndromeU54DK120342 · NIDDK · UNIVERSITY OF CALIFORNIA LOS ANGELES · PI REUE, KAREN · 2018 to 2022
$7.6M
The impact of estrogen receptor alpha on cardiomyocellular metabolism and healthU54HL170326 · NHLBI · UNIVERSITY OF CALIFORNIA LOS ANGELES · PI Hooman Allayee · 2023 to 2026
$7.5M
UC San Diego Medical Scientist Training ProgramT32GM154642 · NIGMS · UNIVERSITY OF CALIFORNIA, SAN DIEGO · PI Neil C Chi, Mary Kathleen Lewinski · 2025 to 2026
$2.9M
Acetylation and contraction-mediated glucose uptakeR21AG067495 · NIA · UNIVERSITY OF CALIFORNIA, SAN DIEGO · PI SCHENK, SIMON · 2021 to 2022
$448k
p300/CBP, acetylation and muscle force generationR21AR072882 · NIAMS · UNIVERSITY OF CALIFORNIA, SAN DIEGO · PI SCHENK, SIMON · 2018 to 2019
Mitochondria are key regulators of metabolism and ATP supply in skeletal muscle, while circadian rhythms influence many physiological processes. However, whether mitochondrial function is intrinsically regulated in a circadian manner in mouse skeletal muscle is inadequately understood. Accordingly, we measured postabsorptive transcript abundance of markers of mitochondrial autophagy, dynamics, and metabolism [extensor digitorum longus (EDL), soleus, gastrocnemius], protein abundance of electron transport chain complexes (EDL and soleus), enzymatic activity of succinate dehydrogenase (tibialis anterior and plantaris), and maximal mitochondrial respiration (tibialis anterior) in different skeletal muscles from female C57BL/6NJ mice at four zeitgeber times: 1, 7, 13, and 19. Our findings demonstrate that markers of mitochondrial function and oxidative metabolism do not display intrinsic time-of-day regulation at the gene, protein, enzymatic, or functional level. The core-clock genes
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
Markers of mitochondrial function and oxidative metabolism in skeletal muscle do not display intrinsic circadian regulation in female mice. · full record | OpenQuestion