Evidence map›Paper›PMID 41051314›Full record

ArticleVirulence2025

Rad53 orchestrates divergent pathways governing genotoxic resistance, morphogenesis, and virulence in

Jia Wang, Shaling Yang, Yi Zhou, Huaxin Cai, Jinrong Feng

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Virulence, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Jia WangDepartment of Pathogen Biology, School of Medicine, Nantong University, Nantong, Jiangsu, China.
Shaling YangDepartment of Pathogen Biology, School of Medicine, Nantong University, Nantong, Jiangsu, China.
Yi ZhouDepartment of Pathogen Biology, School of Medicine, Nantong University, Nantong, Jiangsu, China.
Huaxin CaiDepartment of Pathogen Biology, School of Medicine, Nantong University, Nantong, Jiangsu, China.
Jinrong FengDepartment of Pathogen Biology, School of Medicine, Nantong University, Nantong, Jiangsu, China.ORCID 0000-0002-9444-1100

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The DNA damage response (DDR), an evolutionarily conserved surveillance mechanism enabling cells to detect genomic lesions and cell cycle arrest for repair, coordinates genomic stability through key effector kinases. In

Indexed as

Candida albicansCandidiasisCheckpoint Kinase 2AnimalsDNA DamageDNA RepairDrug Resistance, FungalGene Expression Regulation, FungalHyphaeMaleMiceMice, Inbred ICRPromoter Regions, GeneticRegulatory Factor X1Transcription, GeneticVirulenceCheckpoint Kinase 2Regulatory Factor X1Rfx1 protein, mouseCandida albicanscheckpoint kinasehyphal formationRad53virulence

Identifiers

PMID41051314
PMCPMC12520088

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.