ArticleJACC. Case reports2025
Hypereosinophilic Syndrome Presenting as Coronary Artery Spasm and ST-Segment Elevation Myocardial Infarction.
Article in JACC. Case reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
1 citing paper in PubMed.
- Unmet Need to Verify Coronary Artery Spasm in Patients with Chronic or Acute Coronary Syndrome and Non-Obstructive Coronary Arteries.Life (Basel, Switzerland) · 2026Review
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Authors and funding
4 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
backgroundHypereosinophilic syndrome (HES) is characterized by high absolute eosinophil count and multiorgan damage. Energy drink consumption (EDC) is associated with adverse cardiovascular events, including coronary artery spasm (CAS). CASE SUMMARY: A 31-year-old man with asthma and excessive EDC presented with chest pain, hypotension, and inferior ST-segment elevation myocardial infarction with elevated troponin to 1,081 ng/L. Given persistent CAS refractory to coronary vasodilators, he underwent balloon angioplasty of the right coronary artery and first diagonal branch. Despite initial improvement, his absolute eosinophil count and systemic symptoms worsened. Gastrointestinal, pulmonary, and bone marrow biopsies demonstrated eosinophilic infiltration. He achieved remission with corticosteroids. DISCUSSION: ST-segment elevation myocardial infarction from CAS is a rare cardiac manifestation of HES. The multidisciplinary management requires pharmacologic and interventional therapies to reduce inflammation and prevent cardiovascular sequelae. TAKE-HOME MESSAGE: This case highlights the importance of early recognition and treatment HES in patients with unexplained myocardial infarction, particularly when associated with EDC and the absence of atherosclerotic risk factors.
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