Evidence map›Paper›PMID 41030450›Full record

ReviewFrontiers in immunology2025

DKA with negative DM-autoantibodies, complicated by GBS and RESLES: a case report and literature review.

Nan Tian, Yanwen Yang, Qiang Liu, Zeli Ma, Ying Wang, Xuan Li, Xu Wang, Yanzi Jin, Rong Chen, Yajun Li and 3 more

Abstract readCase ReportsReview
In one paragraph

Review in Frontiers in immunology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Observational
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors.

Nan TianClinical Medical College, Ningxia Medical University, Yinchuan Ningxia, China.
Yanwen YangDepartment of Neurology, General Hospital of Ningxia Medical University, Ningxia, Key Laboratory of Cerebrocranial Disease, Incubation Base of National Key Laboratory, Yinchuan, Ningxia, China.
Qiang LiuDepartment of Neurology, General Hospital of Ningxia Medical University, Ningxia, Key Laboratory of Cerebrocranial Disease, Incubation Base of National Key Laboratory, Yinchuan, Ningxia, China.
Zeli MaClinical Medical College, Ningxia Medical University, Yinchuan Ningxia, China.
Ying WangClinical Medical College, Ningxia Medical University, Yinchuan Ningxia, China.
Xuan LiClinical Medical College, Ningxia Medical University, Yinchuan Ningxia, China.
Xu WangDepartment of Neurology, General Hospital of Ningxia Medical University, Ningxia, Key Laboratory of Cerebrocranial Disease, Incubation Base of National Key Laboratory, Yinchuan, Ningxia, China.
Yanzi JinDepartment of Neurology, General Hospital of Ningxia Medical University, Ningxia, Key Laboratory of Cerebrocranial Disease, Incubation Base of National Key Laboratory, Yinchuan, Ningxia, China.
Rong ChenDepartment of Neuroelectrophysiology, Cardiovascular and Cerebrovascular Disease Hospital Branch, General Hospital of Ningxia, Yinchuan, Ningxia, China.
Yajun LiClinical Medical College, Ningxia Medical University, Yinchuan Ningxia, China.
Fenkui SheClinical Medical College, Ningxia Medical University, Yinchuan Ningxia, China.
Fenglin MouClinical Medical College, Ningxia Medical University, Yinchuan Ningxia, China.
Qing ZhangDepartment of Neurology, General Hospital of Ningxia Medical University, Ningxia, Key Laboratory of Cerebrocranial Disease, Incubation Base of National Key Laboratory, Yinchuan, Ningxia, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Guillain-Barré syndrome (GBS) is an autoimmune-mediated disorder characterized by acute, multifocal peripheral neuropathies, typically triggered by infections, autoimmune diseases, or immunosuppressive therapies. In this report, we describe a patient newly diagnosed with diabetes mellitus (DM) and diabetic ketoacidosis (DKA), who presented with negative insulin-related autoantibodies and no identifiable infectious etiology. It is suggested that DKA may independently contribute to the development of GBS. Additionally, DKA can lead to the reversible splenial lesion syndrome (RESLES). We present a case of GBS with atypical features consistent with the acute motor axonal neuropathy (AMAN) variant.

Indexed as

AutoantibodiesDiabetic KetoacidosisGuillain-Barre SyndromeHumansAutoantibodiesautoantibody-negativeDKADMGBSRESLES

Identifiers

PMID41030450
PMCPMC12477141

What OpenQuestion holds

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LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.