ArticleBMC research notes2025
Characterization of plasmid-mediated quinolone resistant genes among uropathogenic Escherichia coli isolates in Bushehr, south of Iran.
Article in BMC research notes, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
objectivesUropathogenic Escherichia coli (UPEC) is important in urinary tract infection (UTI). Quinolones and fluoroquinolones are broad-spectrum antibiotics for the treatment of UTI, although some of UPEC are resistant to these antibiotics. This study aims to investigate phenotypic and genotypic characterization of plasmid-mediated quinolone resistance UPEC isolated from patients with UTI in Bushehr. In this study, 105 isolates of UPEC were identified by standard microbiological tests and the susceptibility pattern of quinolone and fluoroquinolone were determined by the disc diffusion method. PCR was used to check the presence of plasmid-mediated quinolone resistance genes.
resultsPattern of antibiotic susceptibility showed that more than 50% and 75% of isolates were resistant to fluoroquinolones and quinolones, respectively. The frequencies of qnrS and qnrB genes were 49.5% and 23.8%, respectively. Based on analysis, there was a significant relationship between the presence of the qnrB gene and resistance to ciprofloxacin, norfloxacin, ofloxacin and levofloxacin. There was a significant relationship between the presence of qnrS gene and resistance to the tested antibiotics. Our results showed a high level of resistance to fluoroquinolones. Therefore, it is necessary to treat and prevent UTI so that physicians can use alternative antibiotics to treatment of patients based on laboratory results.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.