ArticleBMC cardiovascular disorders2025
Low air-temperatures exposure as a risk factor for coronary artery disease progression - personalized analysis.
Article in BMC cardiovascular disorders, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
1 citing paper in PubMed.
- Winter Temperature and Long-Term Mortality After Coronary Artery Bypass Grafting: A Multicenter Cohort Study.Journal of clinical medicine · 2026Article
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Authors and funding
14 authors.
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No grant is acknowledged in the PubMed record.
Abstract
backgroundCardiovascular morbidity is still an epidemiological problem, especially related to the risk of re-intervention. In addition to classical atherosclerosis risk factors, the influence of climate changes related to ambient temperature exposure has been recently taken into account. The individual approach is indicated as desirable and justified for clinical conclusion drawing. The possible relation between air temperature and coronary artery atherosclerosis progression was the subject of our study.
methodsOut of 224 patients, 146 (100 (68%) males and 46 (32%) females in a mean (standard devation - SD) age of 67.7 ± 7.7 years) underwent percutaneous coronary intervention (PCI) followed by repeated angiography due to angina equivalent symptoms after median (Q1-3) 510 (154-1451) days. Demographical, clinical, and angiographical characteristics, followed by ambient temperature exposure within the time interval between both angiographic evaluations, were interpreted for predictors of coronary artery stenosis progression.
resultsIn the multivariable analysis, mean yearly ambient temperatures (p = 0.018), exposure to extreme low (below - 10
conclusionsThe coronary disease progression was found to be age-related and modulated by ambient temperature exposures. The low ambient temperatures in patients with previous percutaneous interventions may relate to premature significant coronary culprit lesion progression. The exposure to days characterized by warm temperatures may be regarded as protective. The climatic changes represented by mean yearly temperature growth can be regarded as an ominous factor for plaque progression.
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