ArticleFrontiers in pharmacology2025
Occupational toxic encephalopathy due to 1,2-dichloroethane exposure: a case series.
Article in Frontiers in pharmacology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
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Who cites it
3 citing papers in PubMed.
- Acute toxic encephalopathy induced by organic solvent exposure: a case report of diagnostic challenges and occupational health implications.International journal of emergency medicine · 2025Article
- Caffeic acid phenethyl ester alleviates 1,2-dichloroethane-induced toxic cerebral edema: central and peripheral mechanisms.Frontiers in pharmacology · 2025Article
- Inflammatory mechanisms in brain edema from 1,2-dichloroethane poisoning: a review.Frontiers in neurologyReview
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Authors and funding
10 authors.
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Abstract
1,2-Dichloroethane (1,2-DCE) is a commonly used organic solvent in industrial settings. In pharmacokinetic studies using electrochemical techniques, it is widely used as an organic solvent for dissolving drugs and forms a water/1,2-DCE interface. The 1,2-DCE exposure caused by inadequate protection poses a risk of toxicity via inhalation or dermal contact. This case series documents five instances of poisoning resulting from occupational exposure to industrial products containing 1,2-DCE. 1,2-DCE can induce neurological damage, particularly affecting the central nervous system, manifesting as toxic encephalopathy. Clinical manifestations encompass headache, limb convulsions, and coma, often accompanied by increased intracranial pressure. Magnetic resonance imaging aids in the early detection of toxic encephalopathy by revealing extensive cerebral edema and diffuse, symmetrical abnormalities in signal intensity within the bilateral cerebral white matter, basal ganglia, and dentate nucleus. The principal therapeutic strategies encompass the administration of dehydrating agents, glucocorticoids, and hyperbaric oxygen therapy. Patients with mild poisoning can achieve recovery, whereas those with severe poisoning may experience fatal outcomes. Consequently, effective preventative measures must be instituted to minimize exposure to 1,2-DCE in the workplace.
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