ArticleCureus2025
Diagnostic and Management Challenges in Atypical Central Serous Chorioretinopathy Mimicking Vogt-Koyanagi-Harada Disease: A Case Report.
Article in Cureus, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Central serous chorioretinopathy (CSCR) is characterized by serous detachment of the neurosensory retina and is considered part of the pachychoroid spectrum. Vogt-Koyanagi-Harada (VKH) disease, in contrast, is characterized by bilateral granulomatous panuveitis, exudative retinal detachments, and choroidal thickening. As both are primarily diseases of the choroid, the rare clinical overlap between atypical CSCR and VKH disease can complicate diagnosis. We report a case of atypical CSCR mimicking VKH disease, highlighting the diagnostic and treatment challenges. A 50-year-old woman presented with bilateral progressive reduction in vision over three months, accompanied by headaches and tinnitus. Ocular examination revealed bilateral exudative retinal detachment, and she was diagnosed with acute incomplete VKH disease. Despite systemic corticosteroid therapy, her condition worsened, leading to a revised diagnosis of VKH disease with steroid-induced CSCR. Systemic corticosteroids were tapered and discontinued due to CSCR development. Subsequent surgical interventions, including vitreous biopsy, vitrectomy, subretinal fluid drainage, and silicone oil tamponade, were carried out. One month postoperatively, recurrent exudative retinal detachment occurred. With a suspicion of masquerade syndrome, she underwent left eye removal of oil, retinal and choroidal tissue biopsy, subretinal fluid sampling and drainage, endolaser, and silicone oil tamponade. Histopathological examination and biopsies revealed no malignancy, infection, or inflammation. Postoperatively, visual improvement was limited due to chronic exudative retinal detachment. Ultimately, the diagnosis was revised to atypical CSCR based on clinical findings, disease progression, and multimodal imaging. Atypical bullous retinal detachment is a rare manifestation, emphasizing the need to consider this variant in cases of serous retinal detachment. Timely recognition is essential to guide appropriate treatment and minimize visual complications.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.