ArticlePlant signaling & behavior2025
HOS15 together with PWR-HDA9 positively regulates dark-induced senescence in Arabidopsis.
Article in Plant signaling & behavior, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
1 citing paper in PubMed.
- Mutation of BrDAD1 affects flower opening and anther dehiscence by regulating jasmonate-mediated BrMYB108-BrSWEET15 module.TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik · 2026Article
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8 authors.
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Abstract
Senescence is a conserved phenomenon in all living organisms, including plants. The initiation and progression of leaf senescence can be triggered by natural internal factors or induced by external stress conditions. Over the past few decades, several transcriptional regulators, histone deacetylases/acetyltransferases (HDACs/HATs), signaling transduction pathway components, hormonal regulators, and other proteins have been extensively studied and reported to play a role in regulating leaf senescence. However, a deeper molecular understanding of their mechanisms is needed. We recently reported that a WD40-repeat domain protein, HOS15, regulates aging- and dark-induced senescence. Loss-of-function HOS15 mutant plants exhibited a late senescence phenotype with greater chlorophyll content accumulation. The transcript levels of senescence-related (
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