ReviewAdvances in neurobiology2025
The Effects of Exercise on Hippocampus-Based Cognitive Dysfunction in Type 2 Diabetes Mellitus: A Possible Involvement of Hippocampal Monocarboxylate Transporter 2.
Review in Advances in neurobiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Cognitive dysfunction in type 2 diabetes mellitus (T2DM) poses a significant peril not only to compromised human well-being but also to the onset of dementia, Alzheimer's disease, and depression. Given the present research findings utilizing animal models, various potential biochemical mechanisms have been reported for hippocampus-based cognitive dysfunction in T2DM. This chapter focuses on the relationship between cognitive dysfunction in T2DM and dysregulation of the hippocampal astrocyte-neuron lactate shuttle, with specific emphasis on monocarboxylate transporter 2 (MCT2). Furthermore, it provides a summary of the evidence suggesting the potentiality of exercise as a viable therapeutic intervention, encompassing not solely glycemic control but also the amelioration of cognitive dysfunction in T2DM.
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