ArticleJournal of imaging2025
Combined CCTA and Stress CTP for Anatomical and Functional Assessment of Myocardial Bridges.
Article in Journal of imaging, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
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Who cites it
2 citing papers in PubMed.
- Sex differences in CT-FFR of myocardial bridging with or without atherosclerosis: an AI-based quantitative study.Quantitative imaging in medicine and surgery · 2026Article
- Coronary Artery Anomalies and Anatomical Variants: Cross-Sectional Diagnostic Imaging and Clinical Background.Journal of imaging · 2026Review
Corrections and comments
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Authors and funding
6 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Myocardial bridging (MB) is a congenital coronary anomaly whose clinical impact remains controversial. Coronary computed tomography angiography (CCTA) combined with CT myocardial perfusion imaging (CT-MPI) enables a comprehensive anatomical and functional assessment of MB. This study aimed to investigate whether specific high-risk anatomical features of MB are independently associated with myocardial hypoperfusion, using combined CCTA and CT-MPI. We retrospectively analyzed 81 patients with MB showing high-risk anatomical features (depth ≥ 2.0 mm and length ≥ 25 mm) identified by CCTA, all of whom underwent stress dynamic CT-MPI between May 2022 and December 2025. Patients were classified according to the presence or absence of hypoperfusion in MB-related myocardial segments. Clinical and anatomical variables were compared between two groups using non-parametric tests, and multivariable logistic regression was performed to identify independent predictors of hypoperfusion. Among the 81 patients (mean age, 59.3 ± 11.7 years; 54 males), 26 (32.1%) demonstrated perfusion defects. All MBs were located in the left anterior descending artery (LAD). No significant differences were observed in clinical variables between groups. Bridges associated with hypoperfusion were significantly deeper (
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