ArticleIBRO neuroscience reports2025
Potential neurophysiological mechanisms of intermittent theta-burst stimulation to the left prefrontal cortex for post-stroke unilateral neglect: An exploratory transcranial magnetic stimulation - electroencephalography study.
Article in IBRO neuroscience reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
1 citing paper in PubMed.
- Disrupted integration-segregation balance in the intact hemisphere in chronic spatial neglect.Brain structure & function · 2026Article
Corrections and comments
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Authors and funding
6 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Objective: Unilateral neglect (UN) is a common complication after stroke, with limited effective treatment strategies. This study evaluates the therapeutic effects of intermittent theta-burst stimulation (iTBS) on the intact dorsolateral prefrontal cortex (DLPFC) in UN patients and explores the neurophysiological mechanisms. Methods: Twenty UN patients were randomly assigned to an iTBS or sham stimulation group, receiving 10 consecutive days of treatment. Clinical assessments and TMS-EEG recordings were performed before and after treatment. Results: Patients receiving iTBS showed significant improvements in the Line Cancellation Test (p = 0.012). In addition, iTBS led to notable changes in cortical excitability and neural signal propagation. Specifically, the iTBS group exhibited a significant increase in the N100 component (p < 0.001). Time-frequency analysis also revealed a reduction in theta-band power in the affected frontal cortex post-treatment. At the network level, functional connectivity in the iTBS group was significantly enhanced, particularly within interhemispheric frontal and frontoparietal networks during the later stages of the treatment. Conclusion: iTBS effectively alleviates UN symptoms by modulating cortical excitability and enhancing functional connectivity, suggesting it as a promising therapeutic intervention for UN.
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