ArticleiScience2025
FGL2-induced metabolic dysregulation in enteric neural crest cells provides insight into Hirschsprung disease pathogenesis.
Article in iScience, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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16 authors.
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Abstract
Hirschsprung disease (HSCR) is a congenital disorder characterized by the absence of enteric neural crest cells (ENCCs) in aganglionic segments of the intestine. Here, we report that fibrinogen-like protein 2 (FGL2) is upregulated in aganglionic colon tissues from patients with HSCR.
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