ReviewBiological trace element research2026
Selenium and Selenoproteins in Neutrophil Functions.
Review in Biological trace element research, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
3 citing papers in PubMed.
- Selenite Interacting with Thiols Catalytically Releases NO from GSNO, Cleaves Plasmid DNA and Reduces the Hypotensive Effect of GSNO in Rats.Biological trace element research · 2026Article
- Selenium and Liver Steatosis and Fibrosis: Opposing Direct and Steatosis-Mediated Associations in a Large Cohort.JGH open : an open access journal of gastroenterology and hepatology · 2026Article
- Genetics of selenoproteins and selenoprotein metabolism - An overview of current concepts and emerging aspects.Redox biology · 2026Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors.
Funding
Abstract
Neutrophils are innate immune cells, whose activation leads to extensive production of reactive oxygen species (ROS) through the activation of NADPH oxidases (NOXs). ROS plays a pivotal role in modulating neutrophil functions, including phagocytosis, migration, release of neutrophil extracellular traps (NETs), activation of proinflammatory signaling pathways, and apoptosis. Selenium is an essential micronutrient antioxidant that exhibits biological functions through its translational incorporation as the 21st amino acid selenocysteine. With their diverse enzymatic activities, selenium and selenoproteins partake in the modulation of immune cell activities through regulating multiple cellular functions, including redox balance and antioxidant defense. Given the critical role of ROS in neutrophil function, selenium and selenoproteins are likely to modulate neutrophil activities through regulating both redox-dependent and -independent signaling pathways. Here, we review the current understanding of the role of selenium and selenoproteins in regulating neutrophil functions.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.