ArticlebioRxiv : the preprint server for biology2025
Atypical developmental remodeling of dopamine neurons involves AKT-GSK3β signaling and glia activity.
Xiaofan Zhang, Wei Chen, Yayu Wang, Xun Tu, Xiangyu Zhang, Ronald L Davis, Lily Yeh Jan, Yuh Nung Jan
Abstract readPreprint
In one paragraphArticle in bioRxiv : the preprint server for biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
4 · The recordCorrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what moneyAuthors and funding
8 authors.
Xiaofan ZhangDepartment of Physiology and Howard Hughes Medical Institute, University of California at San Francisco; San Francisco, CA 94158, USA.ORCID 0009-0008-6668-0690 Wei ChenDepartment of Physiology and Howard Hughes Medical Institute, University of California at San Francisco; San Francisco, CA 94158, USA.
Yayu WangDepartment of Physiology and Howard Hughes Medical Institute, University of California at San Francisco; San Francisco, CA 94158, USA.
Xun TuDivision of Neurobiology, Department of Molecular and Cell Biology, Helen Wills Neuroscience Institute, Howard Hughes Medical Institute, University of California, Berkeley, CA 94720, USA.
Xiangyu ZhangDepartment of Physiology and Howard Hughes Medical Institute, University of California at San Francisco; San Francisco, CA 94158, USA.
Ronald L DavisDepartment of Neuroscience, UF Scripps Institute for Biomedical Research, Jupiter, FL 33458, USA.
Lily Yeh JanDepartment of Physiology and Howard Hughes Medical Institute, University of California at San Francisco; San Francisco, CA 94158, USA.
Yuh Nung JanDepartment of Physiology and Howard Hughes Medical Institute, University of California at San Francisco; San Francisco, CA 94158, USA.
Funding
Biology of memoryR35NS097224 · NINDS · UNIVERSITY OF FLORIDA · PI DAVIS, RONALD L · 2017 to 2024
$9.1MDendrite morphogenesis, function and regenerationR35NS097227 · NINDS · UNIVERSITY OF CALIFORNIA, SAN FRANCISCO · PI JAN, YUH NUNG · 2017 to 2024
$5.1MThe TMEM16 Family of Ion Channels and Lipid ScramblasesR35NS122110 · NINDS · UNIVERSITY OF CALIFORNIA, SAN FRANCISCO · PI LILY JAN · 2021 to 2026
$3.2MDendrite development, function, degeneration and regenerationR35NS137312 · NINDS · UNIVERSITY OF CALIFORNIA, SAN FRANCISCO · PI YUH NUNG JAN · 2025 to 2026
$1.4MNINDS NIH HHS R35 NS097224NINDS NIH HHS R35 NS097227NINDS NIH HHS R35 NS122110NINDS NIH HHS R35 NS137312
6 · The paper itselfAbstract
Neuronal remodeling is essential for sculpting neural circuits, and its disruption has been implicated in neurodevelopmental and neuropsychiatric disorders. Yet the molecular and cellular diversity of remodeling across neuron types remains incompletely understood. Here, we uncover a distinct remodeling mode in a subtype of
Identifiers
PMID40950227
PMCPMC12425008
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