Evidence map›Paper›PMID 40940754›Full record

ArticleCells2025

The Combination of Ibrutinib with BH3 Mimetics or Dichloroacetate Is Effective in B-CLL.

Joaquín Marco-Brualla, Oscar Gonzalo, Gemma Azaceta, Isabel Izquierdo, Luis Palomera, Martín Villalba, Isabel Marzo, Alberto Anel

Abstract read
In one paragraph

Article in Cells, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Joaquín Marco-BruallaApoptosis, Immunity and Cancer Group, Department of Biochemistry and Molecular and Cell Biology, Aragon Health Research Institute (IIS-Aragon), University of Zaragoza, 50009 Zaragoza, Spain.ORCID 0000-0002-1710-4821
Oscar GonzaloApoptosis, Immunity and Cancer Group, Department of Biochemistry and Molecular and Cell Biology, Aragon Health Research Institute (IIS-Aragon), University of Zaragoza, 50009 Zaragoza, Spain.
Gemma AzacetaHematology Department, Lozano Blesa Clinical Hospital, 50009 Zaragoza, Spain.ORCID 0000-0001-5068-7355
Isabel IzquierdoHematology Department, Lozano Blesa Clinical Hospital, 50009 Zaragoza, Spain.ORCID 0000-0002-3599-4901
Luis PalomeraHematology Department, Lozano Blesa Clinical Hospital, 50009 Zaragoza, Spain.
Martín VillalbaIRMB, University of Montpellier, INSERM, CNRS, Montpellier CHU, 34295 Montpellier, France.ORCID 0000-0002-4385-4888
Isabel MarzoApoptosis, Immunity and Cancer Group, Department of Biochemistry and Molecular and Cell Biology, Aragon Health Research Institute (IIS-Aragon), University of Zaragoza, 50009 Zaragoza, Spain.ORCID 0000-0002-2315-9079
Alberto AnelApoptosis, Immunity and Cancer Group, Department of Biochemistry and Molecular and Cell Biology, Aragon Health Research Institute (IIS-Aragon), University of Zaragoza, 50009 Zaragoza, Spain.ORCID 0000-0002-5175-8394

Funding

Agencia Estatal de Investigación PID2019-105128RB-I00Agencia Estatal de Investigación PID2022-136799OB-I00Gobierno de Aragón B31_20RGobierno de Aragón B31_23R
6 · The paper itself

Abstract

Since its discovery, the BTK inhibitor ibrutinib has redefined the standard treatments for hematological cancers, such as chronic lymphocytic leukemia (CLL). However, concerns exist regarding its secondary effects in humans and its occasional lack of efficacy in certain malignancies. Therefore, combined therapies with ibrutinib have emerged as promising new approaches. In this study, we aimed to explore its therapeutic potential through different approaches. For this purpose, we combined this drug with the BH3 mimetics ABT-199 and ABT-737, which inhibit anti-apoptotic members of the Bcl-2 family, and with the PDK1 inhibitor dichloroacetate (DCA), respectively. As cell models, we used ex vivo samples from patients and also selected the in vitro CLL cell line Mec-1, generating two sub-lines overexpressing Bcl-XL and Mcl-1, a common feature in this cancer. Results demonstrated a synergistic effect for both approaches, in all tumor cells tested, for both cytostatic and cytotoxic effects. Mechanistically, the expression of Bcl-2-family proteins was explored, exhibiting increases in pro-apoptotic, but also in anti-apoptotic, proteins upon ibrutinib treatment and a relative increase in the amount of the pro-apoptotic protein PUMA after treatment with DCA. Our data provides new insights into combined therapies with ibrutinib for CLL, which further expands our knowledge and the potential of this drug for cancer treatment.

Indexed as

AdenineAntineoplastic Combined Chemotherapy ProtocolsDichloroacetic AcidLeukemia, Lymphocytic, Chronic, B-CellPiperidinesApoptosisBiphenyl CompoundsBridged Bicyclo Compounds, HeterocyclicCell Line, TumorDrug SynergismHumansMyeloid Cell Leukemia Sequence 1 ProteinNitrophenolsPiperazinesProto-Oncogene ProteinsProto-Oncogene Proteins c-bcl-2ABT-737AdenineBiphenyl CompoundsBridged Bicyclo Compounds, HeterocyclicDichloroacetic AcidibrutinibMyeloid Cell Leukemia Sequence 1 ProteinNitrophenolsPiperazinesPiperidinesProto-Oncogene ProteinsProto-Oncogene Proteins c-bcl-2PyrazolesSulfonamidesvenetoclaxB-CLLBcl-XLBH3 mimeticsdichloroacetateibrutinibMcl-1

Identifiers

PMID40940754
PMCPMC12427631

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.