ReviewSports medicine and health science2025
Exercise-induced cardioprotection: From endogenous to exogenous mechanisms.
Review in Sports medicine and health science, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
3 citing papers in PubMed.
- Exploring the role of CaMKII in exercise-induced cardioprotection.Human cell · 2026Review
- Mitochondria: the central hub linking exercise to enhanced cardiac function.Frontiers in physiology · 2026Review
- Two-legged medicine: Exercise-mediated health benefits.Sports medicine and health science · 2025Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Background: Acute myocardial infarction (AMI) remains the leading form of cardiovascular morbidity and mortality, while exercise is a preventative and therapeutic countermeasure. The collective benefits of exercise on the heart are called cardioprotection. Exercise-induced cardioprotection encompasses four broad areas: 1) cardiovascular disease (CVD) risk factor improvement, 2) anatomical remodeling of the heart, 3) improved cardiac physiologic function, and 4) mechanisms of exercise preconditioning. Discussion: With respect to the latter area of cardioprotection, research indicates that a few days of moderate intensity aerobic exercise preconditions the heart against cardiac dysrhythmias, ventricular pump dysfunction, and tissue death. The short duration protective timeframe, hours to days after exercise, indicates that the mechanisms are biochemical in nature. Protective mechanisms within exercised hearts include endogenous antioxidant enzymes, better regulation of cytosolic Ca Conclusion: The current review outlines existing knowledge on exercise and factors of cardiac preconditioning, and highlights the avenues for next-step scientific advances to understanding treatments against AMI.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.