Evidence map›Paper›PMID 40927402›Full record

ArticleClinical case reports2025

Congenital Disorder of Glycosylation Following ATP6AP1 Deficiency With Normal Liver Function: A Case Report.

Amirreza Jabbaripour Sarmadian, Babak Abdinia, Kia Seyed Toutounchi, Mohammad Saberi, Shabnam Eskandarzadeh

Abstract read
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Article in Clinical case reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Article
  2. [ATPase HZhonghua gan zang bing za zhi = Zhonghua ganzangbing zazhi = Chinese journal of hepatology · 2026
    Article
  3. Article
4 · The record

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PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Amirreza Jabbaripour SarmadianPediatric Health Research Center Tabriz University of Medical Sciences Tabriz Iran.ORCID https://orcid.org/0000-0001-7677-8458
Babak AbdiniaPediatric Health Research Center Tabriz University of Medical Sciences Tabriz Iran.ORCID https://orcid.org/0000-0002-4375-4991
Kia Seyed ToutounchiFaculty of Medicine Tabriz University of Medical Sciences Tabriz Iran.ORCID https://orcid.org/0000-0002-6782-8342
Mohammad SaberiDepartment of Medical Genetics, School of Medicine Tehran University of Medical Sciences Tehran Iran.
Shabnam EskandarzadehPediatric Health Research Center Tabriz University of Medical Sciences Tabriz Iran.ORCID https://orcid.org/0000-0001-7500-9657

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Congenital disorders of glycosylation (CDG) are a heterogeneous group of inherited metabolic diseases (IMD) characterized by defects in the synthesis and modification of glycoproteins and glycolipids. One of these disorders is ATP6AP1-CDG, a rare X-linked disease with approximately 30 cases reported so far. Symptoms associated with ATP6AP1-CDG include immunodeficiency, liver dysfunction, and neurological manifestations. This report presents the first case of ATP6AP1-CDG in Iran and the Middle East, in a 5-month-old male infant presenting with fever, vomiting, diarrhea, and poor feeding. The patient had a history of similar symptoms at three and 4 months and had been hospitalized with a diagnosis of gastrointestinal (GI) infection. In addition, he had a history of recurrent seizures, which first began at 45 days old, and was treated with phenobarbital. On physical examinations, the patient was lethargic, severely hypotonic with decreased primitive reflexes, and dehydrated with dry mucous membranes and white plaques of candidiasis. There was no tenderness, guarding, or hepatosplenomegaly in the abdominal examination. Laboratory blood tests were requested, which revealed leukocytosis and normal liver and kidney functions, with negative blood, urine, cerebrospinal fluid, and stool cultures for bacterial growth. Considering the history of recurrent infections, idiopathic seizures, suspected immunodeficiency in the patient's deceased sibling and parental consanguinity, primary immunodeficiency was suspected as a possible diagnosis for the patient. Therefore, a panel of immune function tests was requested, all of which were within the normal range. This panel consisted of IgM, IgG, IgA, B-Cell markers (CD19), T-Cell markers (CD3, CD4, and CD8), TRECs, NK-Cell markers (CD16 and CD56), LTT-PHA, LTT-BCG, and CH50. Furthermore, whole exome sequencing (WES) was requested, which revealed a novel hemizygous deletion in the ATP6AP1 gene (NM_001183.6), designated as c.111_116del; p.Ala40_Ala41del (chrX:153657133 TGGCGGC>T, hg19 assembly).

Indexed as

Ac45 proteinATP6AP1case reportcongenital disorders of glycosylationimmunological deficiency syndromesinborn errors of metabolism

Identifiers

PMID40927402
PMCPMC12414802

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