Evidence map›Paper›PMID 40914769›Full record

ArticleOncogene2025

Multicellular tumor-stromal interactions recapitulate aspects of therapeutic response and human oncogenic signaling in a 3D disease model for H3K27M-altered DIPG.

Meenakshi Upreti, Astgik Petrosyan, Matthew E Thornton, Anahit Hovsepyan, G Esteban Fernandez, David S Koos, Stephanie D Byrum, Samuel G Mackintosh, Jacob K Al-Husseini, Tania Porras and 13 more

Abstract read
In one paragraph

Article in Oncogene, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Review
  2. Review
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

23 authors.

Meenakshi UpretiDivision of Neurosurgery, Children's Hospital Los Angeles, Los Angeles, CA, USA. mupreti@chla.usc.edu.ORCID 0009-0004-3727-2410
Astgik PetrosyanThe Saban Research Institute, Children's Hospital Los Angeles, Los Angeles, CA, USA.
Matthew E ThorntonDepartment of Obstetrics & Gynecology, Keck School of Medicine, USC, Los Angeles, CA, USA.ORCID 0000-0002-1083-2703
Anahit HovsepyanThe Saban Research Institute, Children's Hospital Los Angeles, Los Angeles, CA, USA.
G Esteban FernandezThe Saban Research Institute, Children's Hospital Los Angeles, Los Angeles, CA, USA.
David S KoosThe Saban Research Institute, Children's Hospital Los Angeles, Los Angeles, CA, USA.
Stephanie D ByrumDepartment of Biochemistry & Molecular Biology, University of Arkansas for Medical Sciences, Little Rock, AR, USA.
Samuel G MackintoshDepartment of Biochemistry & Molecular Biology, University of Arkansas for Medical Sciences, Little Rock, AR, USA.
Jacob K Al-HusseiniDivision of Neurosurgery, Children's Hospital Los Angeles, Los Angeles, CA, USA.
Tania PorrasCancer & Blood Diseases Institute, Children's Hospital Los Angeles, Los Angeles, CA, USA.
Joseph HaDivision of Neurosurgery, Children's Hospital Los Angeles, Los Angeles, CA, USA.
Alan J TackettDepartment of Biochemistry & Molecular Biology, University of Arkansas for Medical Sciences, Little Rock, AR, USA.
Miqin ZhangClinical Research Division, Fred Hutchinson Cancer Research Center, Seattle, WA, USA.
Malkiat S JohalDepartment of Chemistry, Pomona College, Claremont, CA, USA.
Anat Erdreich-EpsteinKeck School of Medicine of University of Southern California, Los Angeles, CA, USA.
Susan DurhamDivision of Neurosurgery, Children's Hospital Los Angeles, Los Angeles, CA, USA.
Mark D KriegerDivision of Neurosurgery, Children's Hospital Los Angeles, Los Angeles, CA, USA.
Ashley S MargolKeck School of Medicine of University of Southern California, Los Angeles, CA, USA.
Brendan H GrubbsDepartment of Obstetrics & Gynecology, Keck School of Medicine, USC, Los Angeles, CA, USA.
Timothy C ChambersDepartment of Biochemistry & Molecular Biology, University of Arkansas for Medical Sciences, Little Rock, AR, USA.
Shahab AsgharzadehKeck School of Medicine of University of Southern California, Los Angeles, CA, USA.
Rex A MoatsThe Saban Research Institute, Children's Hospital Los Angeles, Los Angeles, CA, USA.
Peter A ChiarelliDivision of Neurosurgery, Children's Hospital Los Angeles, Los Angeles, CA, USA. pchiarelli@chla.usc.edu.

Funding

Supplement for Google cloud build-outR24GM137786 · NIGMS · UNIV OF ARKANSAS FOR MED SCIS · PI Alan Tackett · 2020 to 2026
$15.4M
USC/CHLA Summer Oncology Research Fellowship (SORF) Program for Medical StudentsR25CA225513 · NCI · CHILDREN'S HOSPITAL OF LOS ANGELES · PI ANAT ERDREICH-EPSTEIN, WIJBE MARTIN KAST · 2019 to 2026
$1.7M
Nanoparticle-Enhanced Radiation Therapy for DIPGK08NS125175 · NINDS · CHILDREN'S HOSPITAL OF LOS ANGELES · PI CHIARELLI, PETER · 2023 to 2025
$582k
NCI NIH HHS R25 CA225513NIGMS NIH HHS R24 GM137786NINDS NIH HHS K08 NS125175United States Department of Defense | United States Army | Army Medical Command | Congressionally Directed Medical Research Programs (CDMRP) CDMRP RA210290U.S. Department of Health & Human Services | NIH | National Institute of General Medical Sciences (NIGMS) R24GM137786U.S. Department of Health & Human Services | NIH | National Institute of Neurological Disorders and Stroke (NINDS) K08NS125175-01A1
6 · The paper itself

Abstract

It has become evident from decades of clinical trials that multimodal therapeutic approaches with focus on cell intrinsic and microenvironmental cues are needed to improve understanding and treat the rare, inoperable, and ultimately fatal diffuse intrinsic pontine glioma (DIPG), now categorized as a diffuse midline glioma. In this study we report the development and characterization of an in vitro system utilizing 3D Tumor Tissue Analogs (TTA), designed to replicate the intricate DIPG microenvironment. The innate ability of fluorescently labeled human brain endothelial cells, microglia, and patient-derived DIPG cell lines to self-assemble has been exploited to generate multicellular 3D TTAs that mimic tissue-like microstructures, enabling an in- depth exploration of the spatio-temporal dynamics between neoplastic and stromal cells. The 3D-TTA model recapitulates clinical patterns of DIPG growth, evidenced by resistance to chemotherapy, HDAC and proteasome inhibitors, as well as sensitization to the antibody-activated innate immune microenvironment including complement proteins and surrounding microglia. Multimodal fluorescence imaging platforms integrated with high-throughput omics revealed that alterations in tumor cell motility and growth in the 3D-TTA model compared to tumor cell only spheroids correlated with specific transcriptomic and proteomic changes. STAT3, ITGA5, LGALS1, SOD2, MVP, and CLIC1, associated with microenvironment signaling, DNA replication, and immune regulation, were identified as potential novel targets in the 3D model. The results indicate that the 3D TTA platform developed here represents a powerful tool for preclinical studies, paving the way for identification/validation of tissue specific biomarkers and novel drug targets, thus advancing disease management strategies for DIPG in children.

Indexed as

Brain Stem NeoplasmsGliomaHistonesCell Line, TumorHumansSignal TransductionSpheroids, CellularStromal CellsTumor MicroenvironmentHistones

Identifiers

PMID40914769
PMCPMC12454143

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.