Evidence map›Paper›PMID 40869911›Full record

ArticleGenes2025

Altered Behavior and Neuronal Activity with Paternal

Daniel S Scott, Violeta Zaric, Carol A Tamminga, Ryan K Butler

Abstract read
In one paragraph

Article in Genes, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Daniel S ScottDepartment of Psychiatry, UT Southwestern Medical Center, Dallas, TX 75390, USA.
Violeta ZaricDepartment of Pediatrics, UT Southwestern Medical Center, Dallas, TX 75390, USA.
Carol A TammingaDepartment of Psychiatry, UT Southwestern Medical Center, Dallas, TX 75390, USA.
Ryan K ButlerDepartment of Psychiatry, UT Southwestern Medical Center, Dallas, TX 75390, USA.ORCID 0000-0002-5857-6489

Funding

Foundation for Prader-Willi Research N/A
6 · The paper itself

Abstract

BACKGROUND/

objectivesPrader-Willi Syndrome (PWS) is a neurodevelopmental disease associated with multiple behavioral features, including a prevalence for psychosis. The genetic causes of PWS are well characterized and involve the silencing or deletion of the paternal copy of a region of chromosome 15q11-13. One gene within this region,

methodsIn this study, we assessed the effects of the microdeletion of the paternal copy of

resultsThe results suggest that

conclusionsThese results implicate the targets of

Indexed as

Prader-Willi SyndromePsychotic DisordersRNA, Small NucleolarAnimalsBehavior, AnimalFemaleGene DeletionGyrus CinguliHippocampusHumansMaleMiceMice, Inbred C57BLNeuronsRNA, Small NucleolarSNORD116 RNA, mouseanxietymemoryPrader–Willi syndromepsychosisSnord116

Identifiers

PMID40869911
PMCPMC12385964

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.