Article in Antibiotics (Basel, Switzerland), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Max Roberto Batista AraújoOperational Technical Nucleus, Microbiology, Hermes Pardini Institute, Vespasiano 33200-000, MG, Brazil.ORCID 0000-0002-3293-8496
Jailan da Silva SousaInstitute of Biological Sciences, Federal University of Minas Gerais, Belo Horizonte 31270-901, MG, Brazil.ORCID 0000-0001-5297-7532
Lincoln de Oliveira Sant'AnnaLaboratory of Diphtheria and Corynebacteria of Clinical Relevance, Department of Microbiology, Immunology and Parasitology, Rio de Janeiro State University, Rio de Janeiro 20550-013, RJ, Brazil.ORCID 0000-0002-6933-8961
Tayná do Carmo Sant'Anna CardosoLaboratory of Diphtheria and Corynebacteria of Clinical Relevance, Department of Microbiology, Immunology and Parasitology, Rio de Janeiro State University, Rio de Janeiro 20550-013, RJ, Brazil.ORCID 0009-0005-2975-1144
Amanda Couto Calazans SilvaLaboratory of Diphtheria and Corynebacteria of Clinical Relevance, Department of Microbiology, Immunology and Parasitology, Rio de Janeiro State University, Rio de Janeiro 20550-013, RJ, Brazil.
Siomar de Castro SoaresInstitute of Biological and Natural Sciences, Federal University of Triângulo Mineiro, Uberaba 38025-350, MG, Brazil.ORCID 0000-0001-7299-3724
Bruno Silva AndradeLaboratory of Bioinformatics and Computational Chemistry, Department of Biological Sciences, State University of Southwest of Bahia, Jequié 45083-900, BA, Brazil.ORCID 0000-0002-8031-9454
Louisy Sanches Dos SantosLaboratory of Diphtheria and Corynebacteria of Clinical Relevance, Department of Microbiology, Immunology and Parasitology, Rio de Janeiro State University, Rio de Janeiro 20550-013, RJ, Brazil.ORCID 0000-0002-5303-6395
Vasco Ariston de Carvalho AzevedoInstitute of Biological Sciences, Federal University of Minas Gerais, Belo Horizonte 31270-901, MG, Brazil.ORCID 0000-0002-4775-2280
Funding
Conselho Nacional de Desenvolvimento Científico e Tecnológico - CNPq 311233/2022-8Conselho Nacional de Desenvolvimento Científico e Tecnológico - CNPq 311249/2024-0Coordenação de Aperfeiçoamento de Pessoal de Nível Superior 88887.023263/2024-00Fundação de Amparo à Pesquisa do Estado de Minas Gerais - FAPEMIG APQ-00214-20Fundação de Amparo à Pesquisa do Estado de Minas Gerais - FAPEMIG APQ-01323-15Fundação de Amparo à Pesquisa do Estado do Rio de Janeiro - FAPERJ E-26/202.088/2020Fundação de Amparo à Pesquisa do Estado do Rio de Janeiro - FAPERJ E-26/205.900/2022Fundação de Amparo à Pesquisa do Estado do Rio de Janeiro - FAPERJ E26/211.554/2019
6 · The paper itself
Abstract
background
objectiveswe report, for the first time in Brazil, the detection and phenotypic/genomic characterization of three atoxigenic ST-339 strains isolated from domestic animals, including one with a ciprofloxacin resistance profile linked to double GyrA mutations (S89L, D93G).
methodsspecies identification was performed by MALDI-TOF MS, followed by in vitro antimicrobial susceptibility testing, whole-genome sequencing, and bioinformatic analyses to predict virulence determinants, antimicrobial resistance genes, CRISPR-Cas systems, mobile genetic elements, and in silico structural analysis as well as phylogenetic reconstruction.
resultswhole-genome sequencing confirmed species identity, revealed high genetic similarity, and identified distinct phylogenetic subclades, suggesting potential international dissemination. Genomic analyses showed conserved virulence determinants, such as incomplete pilus clusters, iron acquisition systems, and the
conclusionsthese findings document a little-studied antimicrobial resistance mechanism in zoonotic
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
Quinolone Resistance and Zoonotic Potential of · full record | OpenQuestion