Article in mBio, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
7 authors.
Travis J ChiarelliDepartment of Microbiology and Immunology, Virginia Commonwealth University Medical Center, School of Medicine, Richmond, Virginia, USA.ORCID 0000-0003-1378-9162
Savannah E SanchezDepartment of Microbiology and Immunology, Virginia Commonwealth University Medical Center, School of Medicine, Richmond, Virginia, USA.
Mary Clark H LindDepartment of Microbiology and Immunology, Virginia Commonwealth University Medical Center, School of Medicine, Richmond, Virginia, USA.
Nathaniel S O'BierDepartment of Microbiology and Immunology, Virginia Commonwealth University Medical Center, School of Medicine, Richmond, Virginia, USA.
Curtis B ReadDepartment of Microbiology and Immunology, Virginia Commonwealth University Medical Center, School of Medicine, Richmond, Virginia, USA.
Richard T MarconiDepartment of Microbiology and Immunology, Virginia Commonwealth University Medical Center, School of Medicine, Richmond, Virginia, USA.ORCID 0000-0002-6035-7306
Jason A CarlyonDepartment of Microbiology and Immunology, Virginia Commonwealth University Medical Center, School of Medicine, Richmond, Virginia, USA.ORCID 0000-0003-2778-5066
Funding
Virus Vector Shared ResourceP30CA016059 · NCI · VIRGINIA COMMONWEALTH UNIVERSITY · PI Renato Martins · 1985 to 2026
$51.0M
The roles of Anaplasma phagocytophilum surface proteins in infectionR01AI072683 · NIAID · VIRGINIA COMMONWEALTH UNIVERSITY · PI CARLYON, JASON A · 2007 to 2021
$5.8M
The roles of ceramide and its derivatives in A. phagocytophilum pathogenesisR01AI139072 · NIAID · VIRGINIA COMMONWEALTH UNIVERSITY · PI CHARLES E. CHALFANT, Jason A Carlyon · 2018 to 2026
$5.7M
Orientia tsutsugamushi Ank-host interactions in scrub typhus pathogenesisR01AI167857 · NIAID · VIRGINIA COMMONWEALTH UNIVERSITY · PI Jason A Carlyon · 2022 to 2026
$2.9M
The roles of Anaplasma phagocytophilum surface proteins in infectionR37AI072683 · NIAID · VIRGINIA COMMONWEALTH UNIVERSITY · PI Jason A Carlyon · 2022 to 2026
$2.6M
The roles of Anaplasma phagocytophilum surface proteins in cellular invasionR56AI072683 · NIAID · VIRGINIA COMMONWEALTH UNIVERSITY · PI CARLYON, JASON A · 2012 to 2012
$368k
Anaplasma phagocytophilum exploitation of regulated exocytosis.F32AI183749 · NIAID · VIRGINIA COMMONWEALTH UNIVERSITY · PI Travis Joseph Chiarelli · 2024 to 2026
$228k
American Heart Association 23POST1020758National Institute of Allergy and Infectious Diseases F32 AI83749National Institute of Allergy and Infectious Diseases R01 AI167857, R37 AI072683, R01 AI139072NCI NIH HHS P30 CA016059NIAID NIH HHS F32 AI183749NIAID NIH HHS R01 AI072683NIAID NIH HHS R01 AI139072NIAID NIH HHS R01 AI167857NIAID NIH HHS R37 AI072683NIAID NIH HHS R56 AI072683
6 · The paper itself
Abstract
Pleomorphism is an evolutionary adaptation by which diverse microorganisms maximize their fitness by transitioning between morphologically distinct forms that perform disparate functions in response to the local microenvironment. Cell division is critical for morphotype transition in many pleomorphic bacterial systems.
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
Distinct modes of cell division drive · full record | OpenQuestion