ArticleEnvironment & health (Washington, D.C.)2025
Inhibition of Circadian Rhythm Gene
Article in Environment & health (Washington, D.C.), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
3 citing papers in PubMed.
- PER1-related inflammatory signaling links night shift exposure to lower lung function in coal miners.iScience · 2026Article
- Global Burden of Non-Communicable Diseases from E‑Cigarette Exposure: Trend Analysis and 2050 Projections.Environment & health (Washington, D.C.) · 2026Article
- The Inflammation-Metabolism Axis in Preserved Ratio Impaired Spirometry: Pathogenic Mechanisms and Translational Implications.Journal of inflammation research · 2026Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
8 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Although electronic cigarettes (e-cigarettes) are a substitute for traditional cigarettes, increasing studies indicate that e-cigarettes are unsafe. Here, we first analyzed the constituents of e-cigarette liquid (e-liquid) and e-liquid vaping-produced aerosols (e-aerosols) by gas chromatography quadrupole time-of-flight mass spectrometry (GC/Q-TOF MS) and inductively coupled plasma-MS (ICP-MS), and our result indicated that the components of e-aerosols differed from those of e-liquid. However, there is insufficient evidence on the toxicity of e-aerosols; therefore, an animal study was conducted accordingly. Mice were exposed to e-aerosols for 30, 60, or 90 days within a whole-body exposure chamber equipped with an air quality monitor. Compared with the control, weakened lung function and fiber deposition in murine lungs were observed following e-cigarette exposure. Microcomputed tomography (Micro-CT) images suggested fibrosis-like lesions in the lungs. Mechanistically, the expression of the core circadian rhythm gene,
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.