ArticleCommunications biology2025
Decoding phospho-regulation and flanking regions in autophagy-associated short linear motifs.
Article in Communications biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
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Who cites it
3 citing papers in PubMed.
- MAVISp: A modular structure-based framework for protein variant effects.Protein science : a publication of the Protein Society · 2026Article
- AlphaFold2 SLiM screen for LC3-LIR interactions in autophagy.Autophagy · 2025Article
- Decoding phospho-regulation and flanking regions in autophagy-associated short linear motifs.Communications biology · 2025Article
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Authors and funding
8 authors.
Funding
Abstract
Short Linear Motifs (SLiMs) play a pivotal role in the interactions between intrinsically disordered proteins and their binding partners. SLiMs can undergo regulation through post-translational modifications, including phosphorylation. The flanking regions surrounding the core motifs also exert a crucial role for the interaction. While phosphorylation and flanking regions are known to influence SLiM function, the mechanistic basis of this regulation remains poorly understood. We integrate biomolecular simulations, in silico high-throughput mutational scans, and biophysical experiments to elucidate the phospho-regulation in SLiMs crucial for autophagy, i.e., the LC3 interacting regions (LIRs). We investigate the Optineurin LIR, which perfectly exemplifies a class of LIR with a complex interplay of phosphorylations and flanking regions. Here we show that specific phosphorylation events and flanking residues modulate binding to LC3 at the atomic level, and that disease-associated mutations alter these interactions in the phosphorylated context. Notably, we establish an approach based on Microfluidic Diffusional Sizing to investigate binding affinities of SLiMs to target proteins, complemented by Surface Plasmon Resonance, enabling precise measurements of dissociation constants and kinetics for a selection of variants. Our work provides a versatile toolkit to characterize phospho-regulated SLiMs, advancing the understanding of important cellular processes.
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Registered trials
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