ArticleScientific reports2025
Radial artery calcification is a predictor of aortic stenosis development and progression after initiation of hemodialysis.
Article in Scientific reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
8 authors.
Funding
Abstract
Aortic stenosis (AS) is an important prognostic cardiovascular disease. However, there are few reports investigating the factors contributing to AS progression in patients with hemodialysis (HD). Because human arterial tissue can be easily harvested during arteriovenous fistula (AVF) surgery, we focused on the association between arterial calcification and AS progression. This is the first study with the aimed to establish a link between radial artery calcification (RAC) level and AS progression in patients with end-stage kidney disease (ESKD). All segments of the radial artery were collected during AVF surgery and stained with the Von Kossa stain. Changes in peak flow velocity (ΔVmax) were calculated based on two echocardiographic findings, and the relationship between RAC level and ΔVmax was analyzed. In the univariate analysis, RAC level, baseline peak aortic jet velocity (Vmax), and age were found to contribute to ΔVmax. After adjusting for age, sex, presence of diabetes, and Vmax at HD initiation, RAC level emerged as an independent factor contributing to ΔVmax. In conclusion, a high RAC level may be a predictor of subsequent development and progression of AS after HD initiation. Our findings may help identify this high-risk group and provide targeted healthcare interventions.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.