Evidence map›Paper›PMID 40813448›Full record

ArticleJournal of human genetics2025

Identification of an IL17RC missense variant in a Chinese family with multiple osteochondromas and ankylosing spondylitis.

Yingchun Zheng, Xuewu Wei, Zhongzhi Gan, Mingming Zhang, Zongrui Shen, Shunfei Ma, Yihao Huang, Fei He, Jian Wang, Fu Xiong

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Article in Journal of human genetics, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

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4 · The record

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5 · Who and what money

Authors and funding

10 authors.

Yingchun Zheng *Department of Medical Genetics, School of Basic Medical Sciences, Southern Medical University, Guangzhou, China.
Xuewu Wei *Department of Medical Genetics, School of Basic Medical Sciences, Southern Medical University, Guangzhou, China.
Zhongzhi GanDepartment of Medical Genetics, School of Basic Medical Sciences, Southern Medical University, Guangzhou, China.
Mingming ZhangDepartment of Medical Genetics, School of Basic Medical Sciences, Southern Medical University, Guangzhou, China.
Zongrui ShenDepartment of Medical Genetics, School of Basic Medical Sciences, Southern Medical University, Guangzhou, China.
Shunfei MaDepartment of Medical Genetics, School of Basic Medical Sciences, Southern Medical University, Guangzhou, China.
Yihao HuangDepartment of Medical Genetics, School of Basic Medical Sciences, Southern Medical University, Guangzhou, China.
Fei HeDepartment of Medical Genetics, School of Basic Medical Sciences, Southern Medical University, Guangzhou, China.
Jian WangDepartment of Orthopedic Surgery, Nanfang Hospital, Southern Medical University, Guangzhou, China. digitalphoto@163.com.
Fu XiongDepartment of Medical Genetics, School of Basic Medical Sciences, Southern Medical University, Guangzhou, China. xiongfu@smu.edu.cn.ORCID http://orcid.org/0000-0003-2428-6165

Funding

National Natural Science Foundation of China (National Science Foundation of China) 32170617National Natural Science Foundation of China (National Science Foundation of China) 32370649National Natural Science Foundation of China (National Science Foundation of China) 82302078Natural Science Foundation of Guangdong Province (Guangdong Natural Science Foundation) 2022A1515110438Natural Science Foundation of Guangdong Province (Guangdong Natural Science Foundation) 2024A1515012899
6 · The paper itself

Abstract

Ankylosing spondylitis (AS) is a chronic and progressive inflammatory arthritis involving disorders of both the immune and skeletal systems. Multiple osteochondromas (MO) is a rare skeletal disorder with a variety of clinical manifestations characterized by multiple benign exostoses. Here, we investigate a Chinese family with HLA-B27-negative AS complicated with MO. Whole-exome sequencing (WES) and Sanger sequencing were used to screen and identify the pathogenic gene. In vitro functional analysis was performed, and a pathogenesis-associated interleukin (IL)-17 receptor C (IL17RC) mutation was analyzed to investigate its effect on phenotypes. WES was used to identify a known missense mutation, NM_000127.3:c.1019 G > A(p.Arg340His), in the pathogenic gene EXT1 that is causal for MO. Moreover, a missense mutation, NM_153461.3:c.1067 C > T(p.Thr356Met), in the IL17RC gene was identified as potentially responsible for AS or spondyloarthritis symptoms in this family. In vitro over-expression of mutant IL17RC decreased its expression and increased the expression of IL17RA, consistent with the expression of these two genes in patients. Mechanistically, mutant IL17RC enhanced the activation of the NF-κB pathway. This study increases our understanding of the pathogenesis and progression of these diseases. Our findings broaden the risk factors in non-HLA-B genes associated with the NF-κB pathway in AS.

Indexed as

Exostoses, Multiple HereditaryMutation, MissenseN-AcetylglucosaminyltransferasesReceptors, Interleukin-17Spondylitis, AnkylosingAdultEast Asian PeopleExome SequencingExostosin 1FemaleGenetic Predisposition to DiseaseHumansMalePedigreeExostosin 1N-AcetylglucosaminyltransferasesReceptors, Interleukin-17

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.