ArticleImaging neuroscience (Cambridge, Mass.)2025
Sensory integration processes characterize concussed athletes with balance deficits.
Article in Imaging neuroscience (Cambridge, Mass.), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Impaired postural control constitutes a major symptom after mild traumatic brain injuries (mTBI/sport-related concussions (SRC)). In order to uphold cognition and behavior during pathological situations, individuals may be characterized by neuronal upregulation. Because postural control necessitates the integration of sensory information within somatosensory (/parietal) cortices, we investigated the hypothesis that athletes with ongoing symptoms after SRC are characterized by increased brain activation within these areas in order to compensate for postural deficits. Sixty-six athletes (27 ± 13 years; 50 men, 16 women) participated in the study. Twenty-two concussed athletes reported high post-concussion symptoms (PCS; symptomatic group), and 22 concussed athletes reported low PCS (asymptomatic group). Twenty-two healthy non-concussed athletes served as a control group. Postural control was assessed by a pressure distribution measuring plate during four balance conditions with eyes closed/open whilst either standing on a stable/unstable surface. Brain oxygenation was collected during postural control tasks by functional near-infrared spectroscopy (fNIRS) above pre- and postcentral cortices of both hemispheres. Increased postural sway was found in symptomatic athletes when compared to control athletes' overall conditions as well as during unstable surface conditions. Symptomatic athletes were characterized by increased brain activation within the parietal cortex overall balance conditions and when compared to asymptomatic athletes. Increased brain activation within somatosensory and parietal cortices during postural control indicates that sensory integration processes are upregulated in concussed athletes with persisting symptoms. However, such potentially compensatory processes seem to constitute an ineffective neuronal mechanism as affected athletes cannot countervail post-concussion balance deficits.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.