ArticleNeurogenetics2025
Whole exome sequencing identifies a novel variant causing Neurodegeneration with Brain Iron Accumulation syndrome (NBIA) in a consanguineous Pashtun family.
Article in Neurogenetics, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
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Who cites it
2 citing papers in PubMed.
- Therapeutic Advances in Major NBIA Disorders: Current Strategies and Translational Challenges.Neurology international · 2026Review
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Authors and funding
12 authors.
Funding
Abstract
Neurodegenerative disorders pose a significant public health problem. Among these, neurodegeneration with brain iron accumulation syndrome (NBIA) is particularly challenging because even MRI imaging findings can be subtle. Understanding the genetic basis of NBIA is, therefore, crucial for early diagnosis. This study aims to identify pathogenic mutations underlying NBIA in a consanguineous Pashtun family from Pakistan, using Next Generation Sequencing and protein structure modeling. Whole exome sequencing identified a novel pathogenic variant segregating in the family. Bioinformatics tools were employed for assessing the pathogenicity of the variant and 3D structure modelling of the protein. Following the filtration of variants according to the pedigree structure, we identified a novel homozygous variant (NM_001286611.1:c.1460A > C, p.Lys487Thr) in the REPS1 gene in the proband. Subsequent analysis confirmed the segregation of this variant within the family. Identification of this mutation expands our understanding of NBIA genetics and aids in early diagnosis. Stabilizing energy calculations support the impact of this variant on normal portion functioning.
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