ArticleERJ open research2025
Increased loop gain and upper airway collapsibility among smoking patients with obstructive sleep apnoea.
Article in ERJ open research, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Authors and funding
5 authors.
Funding
Abstract
Background: Smoking is a known risk factor for obstructive sleep apnoea (OSA). However, the specific pathological mechanism linking smoking to OSA remains unclear. This study aims to explore the endotypic traits of OSA among current and former smokers. Methods: We prospectively collected polysomnographic data from 980 patients with an apnoea-hypopnoea index (AHI) ≥15 h Results: Compared with nonsmokers, current smokers were associated with a 7.6 h Conclusions: Smoking is associated with increased upper airway collapsibility and loop gain among patients with OSA. These effects may be reversible following smoking cessation.
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