ArticleJournal of cellular and molecular medicine2025
WDFY4 Promotes the Progression of Atherosclerosis by Regulating Ferroptosis Mediated by the LAPTM5/CDC42/mTOR/4EBP1/SLC7A11 Pathway.
Article in Journal of cellular and molecular medicine, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
3 citing papers in PubMed.
- Article
- Sex differences in cardiac recovery and ventricular gene expression in a rat model of donation after circulatory death.Biology of sex differences · 2026Article
- WDFY4 Promotes the Progression of Atherosclerosis by Regulating Ferroptosis Mediated by the LAPTM5/CDC42/mTOR/4EBP1/SLC7A11 Pathway.Journal of cellular and molecular medicine · 2025Article
Corrections and comments
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Authors and funding
3 authors.
Funding
Abstract
Atherosclerosis (AS) is a prevalent chronic inflammatory cardiovascular disease and leading cause of death worldwide. WD repeat and FYVE domain-containing 4 (WDFY4), a susceptibility gene for autoimmune diseases and reported AS hub gene, is associated with ferroptosis and inflammation. However, the mechanism by which WDFY4 affects atherosclerosis remains unclear. Here, WDFY4 was upregulated in oxidised low-density lipoprotein (ox-LDL)-treated cells and high-fat diet (HFD)-fed ApoE
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Registered trials
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