Evidence map›Paper›PMID 40742477›Full record

ArticleJournal of molecular histology2025

Gemifloxacin ameliorates acetic acid-induced ulcerative colitis via modulation of inflammatory, oxidative, and adhesive biomarkers and histopathological changes in rats.

Farrah Rasool Jaafar, Rana Khairi Attarbashee, Ahmed Rahmah Abu-Raghif, Hayder Ridha-Salman

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Article in Journal of molecular histology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed.

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4 · The record

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PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Farrah Rasool JaafarDepartment of Pharmacology, College of Medicine, Al-Nahrain University, Baghdad, Iraq.
Rana Khairi AttarbasheeDepartment of Dental Basic Sciences, College of Dentistry, University of Mosul, Mosul, Iraq.
Ahmed Rahmah Abu-RaghifDepartment of Pharmacology, College of Medicine, Al-Nahrain University, Baghdad, Iraq.
Hayder Ridha-SalmanCollege of Pharmacy, Al-Mustaqbal University, Hillah, Babylon, 51001, Iraq. hayder80.ridha@gmail.com.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Ulcerative colitis is a Ulcerative colitis is a chronic inflammatory condition characterized by mucosal damage, oxidative stress, and elevated inflammatory mediators, necessitating innovative strategies. While sulfasalazine remains a standard treatment, alternative agents with dual antibacterial and anti-inflammatory effects are of growing interest. The goal of this study is to determine the ameliorative impact of gemifloxacin in comparison to sulfasalazine on inflammatory, oxidative, and histopathological alterations in a rat model of experimentally evoked ulcerative colitis. 40 Albino-Wistar rats were randomly divided into four groups of ten animals. All groups except Group I obtained a single intra-rectal dose of 4% acetic acid (vol/vol). Alternatively, Group I (Sham group) comprised healthy untreated rats who weren't getting any sort of therapy. Group II (control group) had undergone acetic acid-evoked colitis for one week and provided no medications. The rats in groups III (sulfasalazine) and IV (gemifloxacin) were administered 100 mg/kg of sulfasalazine and 50 mg/kg of gemifloxacin orally on a weekly basis, respectively. The histopathological scores of the colonic tissue, together with the following variables. Treatment with both gemifloxacin and sulfasalazine drastically lowered oxidative biomarkers, specifically malondialdehyde (MDA) and myeloperoxidase (MPO), compared to the colitis control group (p < 0.05). Moreover, both drugs significantly mitigated levels inflammatory biomarkers such as tumor necrosis factor alpha (TNF-α), interleukin-1β (IL-1β), and nuclear factor kappa B (NF-κB) while attenuating levels of adhesive molecules like intercellular adhesive molecule (ICAM-1) and E-selectin compared to the colitis control group (p < 0.05). Additionally, they markedly improved the histopathological scores in acetic acid-aggravated-colonic histopathological scores. Gemifloxacin demonstrated remarkable anti-inflammatory, antioxidant, and tissue-protective impacts in a rat prototype of ulcerative colitis with therapeutic outcomes comparable to those of sulfasalazine. These findings support its promise as an adjuvant medication in controlling and managing inflammatory bowel diseases.

Indexed as

BiomarkersColitis, UlcerativeGemifloxacinOxidative StressAcetic AcidAnimalsAnti-Inflammatory AgentsDisease Models, AnimalInflammationMaleRatsRats, WistarSulfasalazineAcetic AcidAnti-Inflammatory AgentsBiomarkersGemifloxacinSulfasalazineAdhesive biomarkersFluoroquinolonesGemifloxacinImmune-mediated diseaseInflammatory bowel diseaseInflammatory cytokinesOxidative indicatorsUlcerative colitis

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.