ArticleCureus2025
Concurrent Superior Mesenteric Artery and Nutcracker Syndromes: A Case Report.
Article in Cureus, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
2 citing papers in PubMed.
- Superior Mesenteric Artery Syndrome Across Adulthood: A Retrospective Clinicoradiological Case Series.Medicina (Kaunas, Lithuania) · 2026Article
- An Overview of Vascular Compression Syndromes and Associations with Autonomic Dysfunction: A Review.Biomedicines · 2026Review
Corrections and comments
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Authors and funding
7 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Superior mesenteric artery syndrome (SMAS) and nutcracker syndrome (NCS) are rare vascular compression disorders caused by entrapment of the third portion of the duodenum and the left renal vein, respectively, between the abdominal aorta and the superior mesenteric artery (SMA). While both conditions share a common pathophysiological mechanism, their simultaneous occurrence remains exceptionally rare. We report the case of a 16-year-old male patient who presented with persistent epigastric pain, nausea, and bilious vomiting. Clinical history revealed recent unquantified weight loss, with a body mass index (BMI) of 17.5 kg/m². Contrast-enhanced computed tomography (CECT) demonstrated a markedly reduced aortomesenteric angle (14.3°) and distance (3.9 mm), consistent with SMAS, along with compression of the left renal vein, upstream dilation, and collateral venous pathways, supporting the diagnosis of concomitant NCS. Due to the severity of duodenal obstruction and failure of conservative management, the patient underwent a transmesocolic anisoperistaltic gastrojejunostomy. Postoperative recovery was uneventful, with complete resolution of gastrointestinal symptoms and satisfactory nutritional improvement at the three-month follow-up. As NCS was asymptomatic, it was managed conservatively with scheduled clinical and radiological surveillance. This case highlights the importance of comprehensive cross-sectional imaging in identifying dual vascular compression syndromes and emphasizes the need for individualized treatment strategies based on symptomatology and anatomical findings.
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Registered trials
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