ReviewEndocrine practice : official journal of the American College of Endocrinology and the American Association of Clinical Endocrinologists2025
Diagnosis and Management of Hypophosphatemic Disorders.
Review in Endocrine practice : official journal of the American College of Endocrinology and the American Association of Clinical Endocrinologists, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
4 citing papers in PubMed.
- Phosphate: An Anion Controlling Metabolic Functions.Nutrients · 2026Review
- Aromatase: a new regulator of phosphate homeostasis.Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research · 2026Article
- Early Dental Manifestations and Multidisciplinary Management of X-Linked Hypophosphatemic Rickets in a Pediatric Patient: A Case Report.Children (Basel, Switzerland) · 2025Article
- Article
Corrections and comments
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Authors and funding
1 author.
Funding
Abstract
In addition to being an essential component of the mineralized skeleton, phosphate plays a role in different physiologic and cellular functions. Several different mineral-regulating hormones, including 1,25-dihydroxyvitamin D, parathyroid hormone, and fibroblast growth factor 23, modulate serum phosphate levels predominantly at the level of the bone, kidney, and intestine. Impaired serum phosphate levels can lead to nonspecific symptoms, and serum phosphate levels are not routinely measured in laboratory analyses; therefore, disorders of hypophosphatemia are often overlooked during patient evaluations. In the past few decades, significant progress has been made in identifying the genetic causes of and novel therapies for hypophosphatemic disorders. In this review, the regulation of phosphate homeostasis, clinical presentation, workup, disorders, and treatment of hypophosphatemia will be discussed.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.