Evidence map›Paper›PMID 40715832›Full record

ArticleJournal of mathematical biology2025

Re-examining the drivers of variation in parasite loads across hosts in the Tallis-Leyton model.

R McVinish

Abstract read
In one paragraph

Article in Journal of mathematical biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

R McVinishSchool of Mathematics and Physics, University of Queensland, Brisbane, Australia. r.mcvinish@uq.edu.au.ORCID http://orcid.org/0000-0002-5168-7012

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The Tallis-Leyton model is a simple model of parasite acquisition where parasites accumulate in the host without affecting the host's mortality, or eliciting any immune reaction from the host. Furthermore, the parasites do not reproduce in the host. We examine how the variability in parasite loads among hosts is affected by the rate of infectious contacts, the distribution of parasite entering the host during infectious contacts, the host's age, and the distribution of parasite lifetimes. Motivated by empirical studies in parasitology, variability is examined in the sense of the Lorenz order and related metrics. Perhaps counterintuitively, increased variability in the distribution of parasite lifetimes is seen to decrease variability in the parasite loads among hosts.

Indexed as

Host-Parasite InteractionsModels, BiologicalParasite LoadParasitic DiseasesAnimalsHumansMathematical ConceptsParasitesAggregationConvex orderGini indexLorenz orderNegative binomial distributionPietra index

Identifiers

PMID40715832
PMCPMC12296923

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.