ArticleBiological trace element research2026
Estimating the Global Burden and Health Risks Associated with Dietary and Cigarette-Related Cadmium Exposure.
Article in Biological trace element research, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers, 1 of them a synthesis that pooled it.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
3 citing papers in PubMed, 1 synthesis or guideline pooled it.
- Exploring cadmium exposure as a contributor to male infertility: insights from the relationship between environmental pollutants and the male reproductive system.Frontiers in endocrinology · 2026Pooled it
- Article
- Mapping the MicroRNA Landscape of Cadmium Exposure: A Bibliometric and Bioinformatics Analysis of Molecular Targets and Regulatory Pathways.International journal of molecular sciences · 2026Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
5 authors.
Funding
Abstract
To quantitatively assess the global burden and health risks associated with dietary and cigarette-related cadmium (Cd) exposure, and to provide a scientific basis for developing public health policies for the prevention and control of global Cd exposure. This study combined the Cd concentration data in foods and cigarettes and their consumption data to assess the Cd exposure levels from these two sources. The toxicity kinetics (TK) model was used to convert dietary Cd exposure into urinary Cd (U-Cd) exposure to further evaluate the burden of cardiovascular disease (CVD) caused by dietary Cd exposure. The risk assessment model of the United States Environmental Protection Agency (USEPA) was adopted to evaluate the cancer and non-cancer risks caused by cigarette-related Cd exposure. The global average daily dietary Cd intake was 2.20 μg/kg bw/day. The daily Cd exposure to cigarettes was 0.0359 μg/kg bw/day. The proportions of the burden of CVD, stroke and coronary heart disease (CHD) explained by dietary Cd intake were 6.08%, 3.34% and 2.74%, respectively. The excess lifetime cancer risk (ELCR) due to cigarette-related Cd exposure was 0.0037, and the non-cancer hazard quotient (NCHQ) was 26.89. Cd exposure in diet and cigarettes significantly affects the global CVD burden and cancer risk. This study emphasizes the importance of reducing Cd exposure in the environment and formulating relevant policies to mitigate health risks, including strengthening Cd control in food and implementing effective and reasonable tobacco control policies.
Indexed as
Identifiers
40705220What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.