Evidence map›Paper›PMID 40703345›Full record

ArticleIn silico pharmacology2025

Pharmacophoric analogs of Savolitinib reveal promising inhibitors of the c-MET receptor tyrosine kinase for drug discovery: in silico investigation.

Lateef Adegboyega Sulaimon, Hameedah Ayanfunke Bamiji, Mercy Eshiet Akpekong, Toyyibat Adelola Asade, Usman Jamila Bashir, Damola Habeeb Adetunji, Ireoluwa Yinka Joel, Rahmat Adetutu Adisa, Titilola Aderonke Samuel, Ashraf Akintayo Akintola

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Article in In silico pharmacology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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4 · The record

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5 · Who and what money

Authors and funding

10 authors.

Lateef Adegboyega SulaimonDepartment of Chemical Sciences, Laboratory of Molecular and Cancer Research, College of Natural and Applied Sciences, Crescent University, Abeokuta, Ogun State Nigeria.
Hameedah Ayanfunke BamijiDepartment of Chemical Sciences, Laboratory of Molecular and Cancer Research, College of Natural and Applied Sciences, Crescent University, Abeokuta, Ogun State Nigeria.
Mercy Eshiet AkpekongDepartment of Chemical Sciences, Laboratory of Molecular and Cancer Research, College of Natural and Applied Sciences, Crescent University, Abeokuta, Ogun State Nigeria.
Toyyibat Adelola AsadeDepartment of Chemical Sciences, Laboratory of Molecular and Cancer Research, College of Natural and Applied Sciences, Crescent University, Abeokuta, Ogun State Nigeria.
Usman Jamila BashirDepartment of Chemical Sciences, Laboratory of Molecular and Cancer Research, College of Natural and Applied Sciences, Crescent University, Abeokuta, Ogun State Nigeria.
Damola Habeeb AdetunjiDepartment of Chemical Sciences, Laboratory of Molecular and Cancer Research, College of Natural and Applied Sciences, Crescent University, Abeokuta, Ogun State Nigeria.
Ireoluwa Yinka JoelDepartment of Biochemistry, Federal University of Agriculture, Makurdi, Benue State Nigeria.
Rahmat Adetutu AdisaDepartment of Biochemistry, Faculty of Basic Medical Sciences, College of Medicine, University of Lagos, Lagos, Nigeria.
Titilola Aderonke SamuelDepartment of Biochemistry, Faculty of Basic Medical Sciences, College of Medicine, University of Lagos, Lagos, Nigeria.
Ashraf Akintayo AkintolaDepartment of Biomedical Convergence Science and Technology, Kyungpook National University, Daegu, South Korea.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The c-MET receptor tyrosine kinase has become a key focus for therapeutic intervention in various cancer types due to its crucial role in driving cancer progression and metastasis. While Savolitinib, a specific inhibitor of c-MET, has demonstrated promise in clinical trials, its limited availability within the body and the risk of developing resistance have prompted a quest for alternative inhibitors. In this research endeavor, we downloaded and screened a range of pharmacophoric analogs inspired by Savolitinib to explore new avenues for inhibiting c-MET. By delving into the molecular structure of Savolitinib and deciphering how it interacts with the c-MET kinase, we aimed to identify critical structural features and functional groups that could be modified to enhance its inhibitory efficacy and pharmacological properties. Employing computational modeling and pharmacokinetic assessments, this research strives to unveil novel analogs that may exhibit superior potency, selectivity, and overall drug-like attributes. Out of the 997 compounds screened, seventeen demonstrated binding energy values comparable to or greater than - 10.2 kcal/mol obtained for Savolitinib during the redocking experiment. Following a thorough evaluation via in silico ADMETox and drug-likeness prediction, two compounds, namely CID_134565115 and CID_153611202, emerged as promising candidates. They not only exhibited favorable binding energy values but also displayed a well-balanced profile concerning stability, drug-likeness and toxicity. In summary, both CID_134565115 and CID_153611202 demonstrated favorable binding energies and distinctive interaction patterns, surpassing the established Savolitinib. Although our computational study highlights the promising potential of these lead-like candidates in obstructing oncogenic c-MET signaling, further experimental investigations are necessary to verify and establish their preclinical relevance.

Indexed as

c-MET-driven cancersc-MET receptor tyrosine kinaseDrug discoveryPharmacophoric analogsSavolinitib

Identifiers

PMID40703345
PMCPMC12279628

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