Article in Proceedings of the National Academy of Sciences of the United States of America, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Yi-Chun HuangDepartment of Biochemistry and Molecular Biology, Tulane University School of Medicine, Tulane Cancer Center, New Orleans, LA 70112.ORCID 0000-0001-6409-485X
Caique Almeida Machado CostaDepartment of Biochemistry and Molecular Biology, Tulane University School of Medicine, Tulane Cancer Center, New Orleans, LA 70112.ORCID 0000-0002-2229-1000
Nicolas Vergara RuizWeill Institute for Cell and Molecular Biology, Department of Molecular Biology and Genetics, Cornell University, Ithaca, NY 14853.
Xianfeng WangDepartment of Biochemistry and Molecular Biology, Tulane University School of Medicine, Tulane Cancer Center, New Orleans, LA 70112.ORCID 0000-0001-7207-7865
Allison JevittDepartment of Biochemistry and Molecular Biology, Tulane University School of Medicine, Tulane Cancer Center, New Orleans, LA 70112.
Christina Marie BrenemanWeill Institute for Cell and Molecular Biology, Department of Molecular Biology and Genetics, Cornell University, Ithaca, NY 14853.ORCID 0009-0009-7593-7184
Chun HanWeill Institute for Cell and Molecular Biology, Department of Molecular Biology and Genetics, Cornell University, Ithaca, NY 14853.ORCID 0000-0001-7319-8095
Wu-Min DengDepartment of Biochemistry and Molecular Biology, Tulane University School of Medicine, Tulane Cancer Center, New Orleans, LA 70112.ORCID 0000-0002-9098-9769
Funding
Notch signaling and germline-soma interactions in the Drosophila ovarian modelR01GM072562 · NIGMS · TULANE UNIVERSITY OF LOUISIANA · PI DENG, WU-MIN · 2006 to 2025
$5.6M
MAGIC tools for genome-wide mosaic analysis with existing Drosophila resources (Equipment Supplement 2023)R24OD031953 · OD · CORNELL UNIVERSITY · PI HAN, CHUN · 2022 to 2025
$3.1M
Tissue Microenvironment ant Tumor Hotspots in DrosophilaR01CA227789 · NCI · TULANE UNIVERSITY OF LOUISIANA · PI DENG, WU-MIN · 2019 to 2023
$1.7M
Mechanistic investigation of malignant rhabdoid childhood tumor using the Drosophila modelR01CA224381 · NCI · TULANE UNIVERSITY OF LOUISIANA · PI DENG, WU-MIN · 2018 to 2022
$1.7M
Polyploidy and Sex Dimorphism in a Drosophila Tumor ModelR01CA287524 · NCI · TULANE UNIVERSITY OF LOUISIANA · PI Wu-Min Deng · 2024 to 2026
$1.7M
Mechanisms of Natural Phosphatidylserine Exposure in the Nervous SystemR21NS127052 · NINDS · CORNELL UNIVERSITY · PI HAN, CHUN · 2022 to 2023
$430k
HHS | NIH | National Cancer Institute (NCI) R01CA224381HHS | NIH | National Cancer Institute (NCI) R01CA227789HHS | NIH | National Institute of General Medical Sciences (NIGMS) R01GM072562HHS | NIH | National Institute of Neurological Disorders and Stroke (NINDS) R21NS127052HHS | NIH | NIH Office of the Director (OD) R24OD031953NCI NIH HHS R01 CA224381NCI NIH HHS R01 CA227789NCI NIH HHS R01 CA287524NIGMS NIH HHS R01 GM072562NIH HHS R24 OD031953NINDS NIH HHS R21 NS127052NSF | BIO | Division of Integrative Organismal Systems (IOS) IOS-155790
6 · The paper itself
Abstract
Removal of dead and damaged cells is critical for organismal health. Under stress conditions such as nutritional deprivation, infection, or temperature shift, the clearance of nonessential cells becomes a universal strategy to conserve energy and maintain tissue homeostasis. Typically, this task is performed by professional phagocytes such as macrophages. However, nonprofessional phagocytes (NPPs) can also adopt a phagocytic fate under specific circumstances. Similar to professional phagocytes, NPPs undergo transitions from immature to mature states and activation, but the precise cellular and molecular mechanisms governing their maturation, induction, and phagocytic execution remain largely unknown. A notable example of stress-induced phagocytosis is the removal of germline cells by follicle cell-derived NPPs during oogenesis in
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.
Polyploidy promotes transformation of epithelial cells into nonprofessional phagocytes. · full record | OpenQuestion