SynthesisThe journal of international advanced otology2025
Association Between Hematological Indices of Inflammation and Pathogenesis and Prognosis of Sudden Sensorineural Hearing Loss: A Systematic Review and Meta-Analysis.
Synthesis in The journal of international advanced otology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
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Who cites it
4 citing papers in PubMed.
- Audiogram Configuration Predicts Treatment Response in Sudden Sensorineural Hearing Loss.Otolaryngology--head and neck surgery : official journal of American Academy of Otolaryngology-Head and Neck Surgery · 2026Article
- Inflammation and Oxidative-Stress Pathways Are Associated with Idiopathic Sudden Hearing Loss: A Genome-Wide Association Study in 15,494 Japanese Individuals.International journal of molecular sciences · 2026Article
- Association between baseline neutrophil-to-lymphocyte ratio and short-term hearing recovery after glucocorticoid therapy in idiopathic sudden sensorineural hearing loss.Frontiers in medicine · 2026Article
- Serum ENA-78, CXCL1, and caspase-3 are associated with disease severity and early hearing recovery in idiopathic sudden sensorineural hearing loss.Frontiers in neuroscience · 2026Article
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Authors and funding
3 authors.
Funding
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Abstract
Chronic inflammation has been proposed as a contributing factor in the pathogenesis of sudden sensorineural hearing loss (SSNHL). The neutrophil-to-lymphocyte ratio (NLR), platelet-to-lymphocyte ratio (PLR), and mean platelet volume (MPV) are well-established indices of inflammation, but their roles in SSNHL pathogenesis and prognosis remain controversial. This meta-analysis aimed to explore these associations. A systematic review was performed by 2 independent investigators across PubMed, Web of Science, Cochrane Library, and Embase databases. The quality of included studies was assessed using the Newcastle-Ottawa Scale. Twenty-six studies including 2392 SSNHL patients and 1865 controls were included. Recovery data were available for 1343 patients, of whom 785 recovered and 558 did not. Meta-analysis revealed that both NLR (standardized mean difference [SMD]=1.082, 95% CI=0.949, 1.216, P < .001) and PLR (SMD=0.516, 95% CI=0.333, 0.700, P < .001) were significantly elevated in SSNHL patients compared to controls. MPV (SMD=1.103, 95% CI=0.820, 1.483, P=.516) showed no significant difference. Additionally, higher NLR (SMD=-0.472, 95% CI=-0.784, -0.160, P=.003) and PLR (SMD=-0.327, 95% CI=-0.630, -0.023, P=.035) were associated with non-recovery in SSNHL, whereas differences in MPV (SMD=-0.352, 95% CI=-0.881, 0.177, P=.192) were not statistically significant. NLR and PLR may be involved in the pathogenesis of SSNHL and may serve as cost-effective and accessible prognostic markers.
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