ArticleCureus2025
Identifying Aspirin as a Contributing Factor to Persistent Gout: A Case Report.
Article in Cureus, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Gout is a metabolic disorder characterized by acute and chronic inflammatory responses due to monosodium urate (MSU) crystal deposition, commonly affecting the first metatarsophalangeal joint, hands, and/or knees. While this condition is typically self-limiting, recurrent cases can suggest an underlying contributor. Low-dose aspirin is commonly used as a cardiovascular prophylaxis. Although research is limited, aspirin has been implicated in decreased uric acid excretion, thus potentially exacerbating gout symptoms in patients. This case highlights a 67-year-old African American male who presented to a clinic complaining of gout in his right hand. Despite following strict lifestyle practices and medication compliance, pharmacological therapies were rendered ineffective until aspirin was identified as a contributing factor to his gout. Optimizing his medication regimen resolved his gout attacks, which stresses the concept that clinicians must be diligent with lesser-known side effects of medication to improve patient outcomes.
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