ArticleThe oncologist2025
Sporadic medullary thyroid carcinoma with a rare RET transmembrane domain mutation (A641R) that responds to selpercatinib.
Naoki Fukuda, Kazuhisa Toda, Tomohiro Chiba, Ippei Fukada, Seiichi Mori, Akiko Tonooka, Erisa Toda, Jumpei Yoshida, Xiaofei Wang, Ryosuke Oki and 8 more
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18 authors.
Naoki FukudaDepartment of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.ORCID 0000-0002-5475-9309 Kazuhisa TodaDepartment of Head and Neck Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.
Tomohiro ChibaDepartment of Pathology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.ORCID 0000-0003-4374-9997 Ippei FukadaDepartment of Genomic Medicine, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.ORCID 0000-0001-7272-3708 Seiichi MoriDepartment of Genomic Medicine, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.ORCID 0000-0001-8569-4504 Akiko TonookaDepartment of Pathology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.
Erisa TodaDepartment of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.
Jumpei YoshidaDepartment of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.ORCID 0000-0001-5310-3423 Xiaofei WangDepartment of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.
Ryosuke OkiDepartment of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.
Takehiro NakaoDepartment of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.ORCID 0009-0008-4421-0997 Tetsuya UrasakiDepartment of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.
Kenji NakanoDepartment of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.ORCID 0000-0002-3721-004X Makiko OnoDepartment of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.ORCID 0000-0002-3151-451X Junichi TomomatsuDepartment of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.
Kengo TakeuchiDepartment of Pathology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.ORCID 0000-0002-1599-5800 Shunji TakahashiDepartment of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.ORCID 0000-0002-5031-1953 Yuji MiuraDepartment of Medical Oncology, The Cancer Institute Hospital of Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.ORCID 0000-0002-7091-628X Funding
No grant is acknowledged in the PubMed record.
6 · The paper itselfAbstract
Medullary thyroid carcinoma is a rare thyroid malignancy derived from parafollicular C cells that is frequently driven by activating mutations in the REarranged during Transfection (RET) proto-oncogene. While most actionable RET mutations are located in the extracellular cysteine-rich or intracellular tyrosine kinase domains, mutations in the transmembrane domain are exceedingly rare and their oncogenic significance remains unclear. We report a case of a 59-year-old male with sporadic medullary thyroid carcinoma harboring a rare RET A641R mutation in the transmembrane domain. The patient experienced multiple locoregional recurrences after four surgical resections. While the companion diagnostic test did not identify RET mutations, comprehensive genomic profiling using a next-generation sequencing panel revealed the RET A641R mutation. Following administration of selpercatinib, a selective RET inhibitor, a rapid biochemical response with decreased serum carcinoembryonic antigen and calcitonin levels was observed, and radiological assessment showed partial response. This is the first report demonstrating the clinical efficacy of selpercatinib in a patient with medullary thyroid carcinoma harboring a RET A641R mutation, supporting the oncogenic potential of this rare variant. This case also emphasizes the importance of comprehensive genomic profiling in identifying rare but actionable RET alterations that are undetectable by targeted sequencing companion diagnostic tests. Selpercatinib may represent an effective therapeutic option for patients with medullary thyroid carcinoma driven by uncommon RET mutations, including mutations in the transmembrane domain.
Indexed as
Carcinoma, NeuroendocrineProto-Oncogene Proteins c-retPyrazolesPyridinesThyroid NeoplasmsHumansMaleMiddle AgedMutationProto-Oncogene MasMAS1 protein, humanProto-Oncogene MasProto-Oncogene Proteins c-retPyrazolesPyridinesRET protein, humanselpercatinibmedullary thyroid cancerprotein-tyrosine kinaseproto-oncogeneRETselpercatinib
Identifiers
PMID40638225
PMCPMC12404292
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