Evidence map›Paper›PMID 40637098›Full record

ArticleEpilepsia open2025

Drosophila melanogaster as a rapid in vivo assay system for preclinical anti-seizure medication testing.

Emma V Töfflinger, Konstantin L Makridis, M-Marcel Heim, David Owald, Angela M Kaindl

Abstract read
In one paragraph

Article in Epilepsia open, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

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0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Emma V TöfflingerDepartment of Pediatric Neurology, Charité - Universitätsmedizin Berlin, Berlin, Germany.
Konstantin L MakridisDepartment of Pediatric Neurology, Charité - Universitätsmedizin Berlin, Berlin, Germany.ORCID https://orcid.org/0000-0002-2609-4557
M-Marcel HeimInstitute of Neurophysiology, Charité - Universitätsmedizin Berlin, Berlin, Germany.ORCID https://orcid.org/0000-0002-8079-4204
David OwaldInstitute of Neurophysiology, Charité - Universitätsmedizin Berlin, Berlin, Germany.ORCID https://orcid.org/0000-0001-7747-7884
Angela M KaindlDepartment of Pediatric Neurology, Charité - Universitätsmedizin Berlin, Berlin, Germany.ORCID https://orcid.org/0000-0001-9454-206X

Funding

Bundesministerium für Bildung und ForschungGünter Endres Fond through the Einstein FoundationSonnenfeld Stiftung
6 · The paper itself

Abstract

Epilepsy represents a significant medical challenge, with a third of patients failing to achieve seizure freedom despite the use of multiple different anti-seizure medications (ASM). Drug resistance is common in genetically caused epilepsies. Patients are faced with repeated, long-lasting, and frequently frustrating drug testing, necessitating targeted therapies and drug repurposing. While in silico tools offer some insight, novel and often genetic epilepsies require preclinical models, which are expensive and time-consuming. Here, we propose Drosophila melanogaster as a rapid in vivo model for preclinical ASM efficacy testing using the sodium channel-associated epilepsies, Dravet syndrome (DS), and generalized epilepsy with febrile seizures plus (GEFS+) as model disorders. We utilize vinegar fly models of DS and GEFS+ that exhibit phenotypic similarities to human patients, including seizures and increased morbidity. Moreover, treatment with ASM effective in humans (clobazam, stiripentol, fenfluramine) reduces seizures, while the application of sodium channel blocking ASM (phenytoin) was deleterious, underlining the model's utility. The utilization of Drosophila as a preclinical model offers a promising avenue for studying genetic epilepsies and assessing ASM efficacy. This approach has the potential to facilitate the development of tailored treatments for patients using a rapidly available in vivo model. PLAIN LANGUAGE SUMMARY: Epilepsy is a challenging condition, with about one-third of patients unable to control seizures despite trying multiple drug treatments. This is especially common in genetic epilepsies. Developing new treatments is expensive and slow, highlighting the need for faster, targeted approaches. This study uses the fly (Drosophila melanogaster) as a rapid, cost-effective model to study genetic epilepsies like Dravet syndrome (DS). These fly models mimic key symptoms seen in humans, including seizures and shorter lifespans. Effective human anti-seizure medications (e.g., clobazam, stiripentol, and fenfluramine) reduced seizures, while sodium channel blockers like phenytoin worsened them. The Drosophila model offers a promising and efficient way to study genetic epilepsies and test treatments, accelerating the development of more targeted therapies.

Indexed as

AnticonvulsantsDisease Models, AnimalEpilepsies, MyoclonicEpilepsy, GeneralizedSeizuresAnimalsDrosophila melanogasterDrug Evaluation, PreclinicalHumansSeizures, FebrileAnticonvulsantsanti‐seizure medicationdevelopmental and epileptic encephalopathyDravet syndromeDrosophila melanogasterepilepsy

Identifiers

PMID40637098
PMCPMC12362156

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.