Evidence map›Paper›PMID 40622523›Full record

ArticleMolecular neurobiology2025

Ecdysterone and High-Intensity Interval Training Mitigate Alzheimer's Pathology in Rats: Impacts on Depression, Synaptic Plasticity, and Neuroinflammation.

Parsa Gholipour, Alireza Komaki, Hesam Parsa, Mahdi Ramezani

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Article in Molecular neurobiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Parsa GholipourNeurophysiology Research Center, Hamadan University of Medical Sciences, Hamadan, Iran. parsagholipour1996@gmail.com.
Alireza KomakiNeurophysiology Research Center, Hamadan University of Medical Sciences, Hamadan, Iran.
Hesam ParsaDepartment of Exercise Physiology, Faculty of Sport Sciences, Bu Ali Sina University, Hamedan, Iran.
Mahdi RamezaniDepartment of Anatomy, School of Medicine, Hamadan University of Medical Sciences, Hamadan, Iran. m.ramezani@umsha.ac.ir.

Funding

Vice Chancellor for Research and Technology, Hamadan University of Medical Sciences IR.UMSHA.AEC.1402.020
6 · The paper itself

Abstract

The study focuses on the role of neuroinflammation and impaired synaptic plasticity in the progression of Alzheimer's disease caused by amyloid-beta accumulation. It examines the potential therapeutic effects of Ecdysterone, known for its anti-inflammatory and antioxidant properties, and high-intensity interval training, which may also support brain health. The primary goal is to assess how Ecdy and HIIT, alone or combined, influence depressive-like behavior, synaptic function, inflammation, and Aβ buildup in a rat model of AD. Ten days after Aβ administration, treatments began with Ecdy (10 mg/kg/day, orally) and/or HIIT, continuing for 8 weeks. Rats were tested for depression-like behavior using the forced swim test. Brain synaptic plasticity was measured through long-term potentiation at the perforant path-dentate gyrus synapse by analyzing population spike amplitude and fEPSP slope. Congo red staining was used to identify Aβ plaques in the brain, and neuroinflammatory markers were quantified in both the hippocampus and cerebral cortex. Aβ injection led to depression, impaired synaptic plasticity, increased inflammation, and Aβ buildup in the brain. While Ecdy and HIIT individually offered some protection, their combination was significantly more effective in improving depression, restoring synaptic function, reducing inflammation, and decreasing Aβ accumulation in both the hippocampus and cerebral cortex (0.05 > P). This data provides evidence that HIIT, accompanied by Ecdy, improves Aβ-induced depression-like behavior, which may be partly related to the amelioration of synaptic dysfunction, a decrease in neuroinflammation, and suppression of Aβ plaque formation.

Indexed as

Alzheimer DiseaseDepressionHigh-Intensity Interval TrainingNeuroinflammatory DiseasesNeuronal PlasticityAmyloid beta-PeptidesAnimalsDisease Models, AnimalMaleRatsRats, Sprague-DawleyAmyloid beta-PeptidesAlzheimer’s disease (AD)Amyloid-beta (Aβ)DepressionEcdysterone (Ecdy)High-intensity interval training (HIIT)Long-term potentiation (LTP)Neuroinflammation

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.