ArticleNeural regeneration research2026
Interplay between brain-specific microRNAs and Alzheimer's disease.
Article in Neural regeneration research, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
3 citing papers in PubMed.
- Mitochondria-associated endoplasmic reticulum membranes and calcium ion exchange: A novel direction for aging and neurodegenerative diseases.Neural regeneration research · 2026Article
- Neuromodulation techniques targeting neurotransmitter dysfunction: Innovation in treatments for Alzheimer's disease.Neural regeneration research · 2026Article
- Aberrant histone acetylation and dysregulated synaptic plasticity in cognitive impairment induced by a high-methionine diet.Neural regeneration research · 2026Article
Corrections and comments
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Authors and funding
5 authors.
Funding
Abstract
Alzheimer's disease is a progressive neurodegenerative disease characterized by memory decline and the accumulation of abnormal protein aggregates in the brain. While the precise cause of Alzheimer's disease remains under investigation, recent research suggests that dysregulation of brain-specific microRNAs (miRs) plays a significant role in Alzheimer's disease pathogenesis. Brain-specific miRs are predominantly expressed within the central nervous system and are crucial for neuronal development, and function, potentially in brain disorders. This review identifies some key brain-specific miRs in Alzheimer's disease, including miR-9, miR-26b, miR-34a, miR-107, miR-124, miR-125b, miR-128, miR-132, miR-146a, miR-155, miR-219, miR-501-3p, and miR-502-3p. The review also shed light on the brain-specific location of these miRs, their dysregulation in Alzheimer's disease, and how they are involved in disease progression. Apparently, these brain-specific miRs modulate specific genes and are therefore crucial for various cellular processes, including autophagy, cell cycle, tau phosphorylation, amyloid-beta production, and neuroinflammation. Moreover, these miRs are potent disease-modifying factors and their expression levels could serve as potential biomarkers for diagnosing or monitoring Alzheimer's disease progression.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.